Evidence that NOS2 acts as a trigger and mediator of late preconditioning induced by acute systemic hypoxia

Chronic systemic hypoxia (SH) enhances myocardial ischemic tolerance in mammals. Xi et al studied the delayed cardioprotection caused by acute SH and associated signaling mechanism.

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Veröffentlicht in:American journal of physiology. Heart and circulatory physiology 2002-07, Vol.52 (1), p.H5-H12
Hauptverfasser: LEI XI, TEKIN, Demet, GURSOY, Erdal, SALLOUM, Fadi, LEVASSEUR, Joseph E, KUKREJA, Rakesh C
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container_title American journal of physiology. Heart and circulatory physiology
container_volume 52
creator LEI XI
TEKIN, Demet
GURSOY, Erdal
SALLOUM, Fadi
LEVASSEUR, Joseph E
KUKREJA, Rakesh C
description Chronic systemic hypoxia (SH) enhances myocardial ischemic tolerance in mammals. Xi et al studied the delayed cardioprotection caused by acute SH and associated signaling mechanism.
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1522-1539
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source American Physiological Society Paid; EZB-FREE-00999 freely available EZB journals
subjects Biological and medical sciences
Cardiology. Vascular system
Coronary heart disease
Heart
Heart attacks
Medical sciences
Nitric oxide
Oxygen
title Evidence that NOS2 acts as a trigger and mediator of late preconditioning induced by acute systemic hypoxia
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