Evidence that NOS2 acts as a trigger and mediator of late preconditioning induced by acute systemic hypoxia

Evidence that NOS2 acts as a trigger and mediator of late preconditioning induced by acute systemic hypoxia

Chronic systemic hypoxia (SH) enhances myocardial ischemic tolerance in mammals. Xi et al studied the delayed cardioprotection caused by acute SH and associated signaling mechanism.

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Bibliographische Detailangaben
Veröffentlicht in:American journal of physiology. Heart and circulatory physiology 2002-07, Vol.52 (1), p.H5-H12
Hauptverfasser: LEI XI, TEKIN, Demet, GURSOY, Erdal, SALLOUM, Fadi, LEVASSEUR, Joseph E, KUKREJA, Rakesh C
Format: Artikel
Sprache:eng
Schlagworte:
Biological and medical sciences
Cardiology. Vascular system
Coronary heart disease
Heart
Heart attacks
Medical sciences
Nitric oxide
Oxygen
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Zusammenfassung:Chronic systemic hypoxia (SH) enhances myocardial ischemic tolerance in mammals. Xi et al studied the delayed cardioprotection caused by acute SH and associated signaling mechanism.
ISSN:0363-6135
1522-1539