Neurocognitive and Synaptic Potentiation Deficits Are Mitigated by Inhibition of HIF1a Signaling following Intermittent Hypoxia in Rodents

Neurocognitive and Synaptic Potentiation Deficits Are Mitigated by Inhibition of HIF1a Signaling following Intermittent Hypoxia in Rodents

A HIF1a-Dependent Pro-Oxidant State Disrupts Synaptic Plasticity and Impairs Spatial Memory in Response to Intermittent Hypoxia. Alejandra Arias-Cavieres, Maggie A. Khuu, Chinwendu U. Nwakudu, Jasmine E. Barnard, Gokhan Dalgin and Alfredo J. Garcia III.

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Bibliographische Detailangaben
Veröffentlicht in:eNeuro 2020-11, Vol.7 (6), p.ENEURO.0449-20.2020, Article 0449
1. Verfasser: Carney, Rosalind S. E.
Format: Artikel
Sprache:eng
Schlagworte:
Animals
Hippocampus
Hypoxia
Life Sciences & Biomedicine
Neuronal Plasticity
Neurosciences
Neurosciences & Neurology
Reactive Oxygen Species
Rodentia
Science & Technology
Spatial Memory
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Zusammenfassung:A HIF1a-Dependent Pro-Oxidant State Disrupts Synaptic Plasticity and Impairs Spatial Memory in Response to Intermittent Hypoxia. Alejandra Arias-Cavieres, Maggie A. Khuu, Chinwendu U. Nwakudu, Jasmine E. Barnard, Gokhan Dalgin and Alfredo J. Garcia III.
ISSN:2373-2822
2373-2822
DOI:10.1523/ENEURO.0449-20.2020