Terminal differentiation into adipocyte and growth inhibition by PPARγ activation in human A549 lung adenocarcinoma cells

The present study investigated the terminal differentiation capacity into adipocytes and subsequent growth inhibition in A549 cancer cells treated with pioglitazone (PGZ), a PPARγ activator. The rate of cell growth in A549 cells was significantly (P

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Bibliographische Detailangaben
Veröffentlicht in:	Animal cells and systems 2020, 24(6), , pp.329-340
Hauptverfasser:	Kim, Dae-Young, Moon, Sun-Ha, Han, Jang-Ho, Kim, Mi-Jeong, Oh, Seong-Ju, Bharti, Dinesh, Lee, Sung-Ho, Park, Jong-Kuen, Rho, Gyu-Jin, Jeon, Byeong-Gyun
Format:	Artikel
Sprache:	eng
Schlagworte:	A549 cells Adenocarcinoma adipocyte Adipocytes Cancer Cell Biology Cell cycle Cell differentiation Cell growth Cell size Chemotherapy Cytotoxicity Developmental Biology differentiation Differentiation (biology) Exposure Fibroblasts G1 phase Galactosidase Glucose metabolism Growth inhibition Human Life Sciences & Biomedicine Pioglitazone Science & Technology Senescence Telomerase Vesicles Zoology β-Galactosidase 생물학
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Beschreibung
Zusammenfassung:	The present study investigated the terminal differentiation capacity into adipocytes and subsequent growth inhibition in A549 cancer cells treated with pioglitazone (PGZ), a PPARγ activator. The rate of cell growth in A549 cells was significantly (P
ISSN:	1976-8354 2151-2485
DOI:	10.1080/19768354.2020.1847731