Happy Hypoxemia in COVID-19A Neural Hypothesis

Many COVID-19 patients are presenting with atypical clinical features. Happy hypoxemia with almost normal breathing, anosmia in the absence of rhinitis or nasal obstruction, and ageusia are some of the reported atypical clinical findings. Based on the clinical manifestations of the disease, we are p...

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Veröffentlicht in:	ACS chemical neuroscience 2020-07, Vol.11 (13), p.1865-1867
Hauptverfasser:	U.R, Anoop, Verma, Kavita
Format:	Artikel
Sprache:	eng
Schlagworte:	Betacoronavirus Biochemistry & Molecular Biology Chemistry, Medicinal Coronavirus Infections - complications Coronavirus Infections - physiopathology COVID-19 Cranial Nerves - physiopathology Cranial Nerves - virology Humans Hypoxia - etiology Hypoxia - physiopathology Inflammation - etiology Inflammation - physiopathology Inflammation - virology Life Sciences & Biomedicine Neurosciences Neurosciences & Neurology Pandemics Pharmacology & Pharmacy Pneumonia, Viral - complications Pneumonia, Viral - physiopathology SARS-CoV-2 Science & Technology Solitary Nucleus - physiopathology Solitary Nucleus - virology
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Beschreibung
Zusammenfassung:	Many COVID-19 patients are presenting with atypical clinical features. Happy hypoxemia with almost normal breathing, anosmia in the absence of rhinitis or nasal obstruction, and ageusia are some of the reported atypical clinical findings. Based on the clinical manifestations of the disease, we are proposing a new hypothesis that SARS-CoV-2 mediated inflammation of the nucleus tractus solitarius may be the reason for happy hypoxemia in COVID-19 patients.
ISSN:	1948-7193 1948-7193
DOI:	10.1021/acschemneuro.0c00318