The Mechanism of Acute Lung Injury Induced by Nickel Carbonyl in Rats
Nickel carbonyl is a highly toxic metal compound produced from the reaction that occurs between nickel and carbon monoxide under pressure. As previously reported, nickel carbonyl can cause acute aspiration pneumonia, and animal experiments showed it was toxic to animal lung, liver, brain, and other...
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| Veröffentlicht in: | Biomedical and environmental sciences 2013-07, Vol.26 (7), p.625-628 |
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| container_title | Biomedical and environmental sciences |
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| creator | BAI, Ya Na MA, Li WANG, Qiu Ying PU, Hong Quan ZHANG, Xiao Pei WU, Xi Jiang XUAN, Xiao Qiang CHENG, Ning |
| description | Nickel carbonyl is a highly toxic metal compound produced from the reaction that occurs between nickel and carbon monoxide under pressure. As previously reported, nickel carbonyl can cause acute aspiration pneumonia, and animal experiments showed it was toxic to animal lung, liver, brain, and other vital organs[1]. However, few studies have investigated nickel carbonyl poisoning in humans. |
| doi_str_mv | 10.3967/0895-3988.2013.07.018 |
| format | Article |
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As previously reported, nickel carbonyl can cause acute aspiration pneumonia, and animal experiments showed it was toxic to animal lung, liver, brain, and other vital organs[1]. However, few studies have investigated nickel carbonyl poisoning in humans.</description><identifier>ISSN: 0895-3988</identifier><identifier>EISSN: 2214-0190</identifier><identifier>DOI: 10.3967/0895-3988.2013.07.018</identifier><identifier>PMID: 23895712</identifier><language>eng</language><publisher>Netherlands: Elsevier B.V</publisher><subject>Acute Lung Injury - chemically induced ; Acute Lung Injury - metabolism ; Acute Lung Injury - pathology ; Animals ; CDC2 Protein Kinase - genetics ; CDC2 Protein Kinase - metabolism ; Cell Cycle - drug effects ; Checkpoint Kinase 1 ; Female ; Lung - metabolism ; Lung - pathology ; Lung - ultrastructure ; Male ; Malondialdehyde - metabolism ; Microscopy, Electron, Transmission ; Organometallic Compounds - toxicity ; Oxidative Stress ; Protein Kinases - genetics ; Protein Kinases - metabolism ; Rats ; Rats, Sprague-Dawley ; RNA, Messenger - metabolism ; 一氧化碳 ; 动物实验 ; 大鼠 ; 急性 ; 损伤机制 ; 羰基镍 ; 肺炎 ; 金属化合物</subject><ispartof>Biomedical and environmental sciences, 2013-07, Vol.26 (7), p.625-628</ispartof><rights>2013 The Editorial Board of Biomedical and Environmental Sciences</rights><rights>Copyright © Wanfang Data Co. 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All Rights Reserved.</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Uhttp://image.cqvip.com/vip1000/qk/84046X/84046X.jpg</thumbnail><linktohtml>$$Uhttps://dx.doi.org/10.3967/0895-3988.2013.07.018$$EHTML$$P50$$Gelsevier$$H</linktohtml><link.rule.ids>314,776,780,3536,27903,27904,45974</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/23895712$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>BAI, Ya Na</creatorcontrib><creatorcontrib>MA, Li</creatorcontrib><creatorcontrib>WANG, Qiu Ying</creatorcontrib><creatorcontrib>PU, Hong Quan</creatorcontrib><creatorcontrib>ZHANG, Xiao Pei</creatorcontrib><creatorcontrib>WU, Xi Jiang</creatorcontrib><creatorcontrib>XUAN, Xiao Qiang</creatorcontrib><creatorcontrib>CHENG, Ning</creatorcontrib><title>The Mechanism of Acute Lung Injury Induced by Nickel Carbonyl in Rats</title><title>Biomedical and environmental sciences</title><addtitle>Biomedical and Environmental Sciences</addtitle><description>Nickel carbonyl is a highly toxic metal compound produced from the reaction that occurs between nickel and carbon monoxide under pressure. As previously reported, nickel carbonyl can cause acute aspiration pneumonia, and animal experiments showed it was toxic to animal lung, liver, brain, and other vital organs[1]. However, few studies have investigated nickel carbonyl poisoning in humans.</description><subject>Acute Lung Injury - chemically induced</subject><subject>Acute Lung Injury - metabolism</subject><subject>Acute Lung Injury - pathology</subject><subject>Animals</subject><subject>CDC2 Protein Kinase - genetics</subject><subject>CDC2 Protein Kinase - metabolism</subject><subject>Cell Cycle - drug effects</subject><subject>Checkpoint Kinase 1</subject><subject>Female</subject><subject>Lung - metabolism</subject><subject>Lung - pathology</subject><subject>Lung - ultrastructure</subject><subject>Male</subject><subject>Malondialdehyde - metabolism</subject><subject>Microscopy, Electron, Transmission</subject><subject>Organometallic Compounds - toxicity</subject><subject>Oxidative Stress</subject><subject>Protein Kinases - genetics</subject><subject>Protein Kinases - metabolism</subject><subject>Rats</subject><subject>Rats, Sprague-Dawley</subject><subject>RNA, Messenger - metabolism</subject><subject>一氧化碳</subject><subject>动物实验</subject><subject>大鼠</subject><subject>急性</subject><subject>损伤机制</subject><subject>羰基镍</subject><subject>肺炎</subject><subject>金属化合物</subject><issn>0895-3988</issn><issn>2214-0190</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2013</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNo9kU1v1DAQhi0EokvbnwAyEgcu2Y4_ktinqloVqLRQqWrPlu1Mdr3NOm2cgPbf47Atp5HtRzPvPCbkI4Ol0FV9AUqXhdBKLTkwsYR6CUy9IQvOmSyAaXhLFv-ZE_IhpR2AZFqq9-SEi_xSM74g1_dbpD_Rb20MaU_7ll75aUS6nuKG3sTdNBxyaSaPDXUH-iv4R-zoyg6uj4eOhkjv7JjOyLvWdgnPX-opefh2fb_6Uaxvv9-srtaFF5yPBXqUWIOuXKudrhtpW-XzqZIeqlI4q4RrtLeNUiUH0Uq0rdPSSS1EI7kUp-TLse8fG1sbN2bXT0PME43DNHuAOlvI2Ncj9jT0zxOm0exD8th1NmI_JcMkq0DKUrCMfnpBJ7fHxjwNYW-Hg3k1lIHLI4B5r98BB5N8wJh9hAH9aJo-GAZm_hMz-zazbzNnMZBv_qX5fOzgt33cPIcc-3WKrCpdigrEX9i8hf4</recordid><startdate>20130701</startdate><enddate>20130701</enddate><creator>BAI, Ya Na</creator><creator>MA, Li</creator><creator>WANG, Qiu Ying</creator><creator>PU, Hong Quan</creator><creator>ZHANG, Xiao Pei</creator><creator>WU, Xi Jiang</creator><creator>XUAN, Xiao Qiang</creator><creator>CHENG, Ning</creator><general>Elsevier B.V</general><general>School of Public Health, Lanzhou University, Lanzhou 730000, Gansu, China%College of Earth and Environmental Science, Lanzhou University, Lanzhou 730000, Gansu, China%Hospital Workers of Jinchuan Company, Jinchuan Group Co.,Ltd., Jinchuan 737103, Gansu, China%Key Laboratory of Preclinical Study for New Medicine of Gansu Province, College of Basic Medicine, Lanzhou University, Lanzhou 730000, Gansu, China</general><general>College of Earth and Environmental Science, Lanzhou University, Lanzhou 730000, Gansu, China</general><scope>2RA</scope><scope>92L</scope><scope>CQIGP</scope><scope>W91</scope><scope>~WA</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>7X8</scope><scope>2B.</scope><scope>4A8</scope><scope>92I</scope><scope>93N</scope><scope>PSX</scope><scope>TCJ</scope></search><sort><creationdate>20130701</creationdate><title>The Mechanism of Acute Lung Injury Induced by Nickel Carbonyl in Rats</title><author>BAI, Ya Na ; 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As previously reported, nickel carbonyl can cause acute aspiration pneumonia, and animal experiments showed it was toxic to animal lung, liver, brain, and other vital organs[1]. However, few studies have investigated nickel carbonyl poisoning in humans.</abstract><cop>Netherlands</cop><pub>Elsevier B.V</pub><pmid>23895712</pmid><doi>10.3967/0895-3988.2013.07.018</doi><tpages>4</tpages></addata></record> |
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| ispartof | Biomedical and environmental sciences, 2013-07, Vol.26 (7), p.625-628 |
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| source | MEDLINE; Elsevier ScienceDirect Journals; EZB-FREE-00999 freely available EZB journals; Alma/SFX Local Collection |
| subjects | Acute Lung Injury - chemically induced Acute Lung Injury - metabolism Acute Lung Injury - pathology Animals CDC2 Protein Kinase - genetics CDC2 Protein Kinase - metabolism Cell Cycle - drug effects Checkpoint Kinase 1 Female Lung - metabolism Lung - pathology Lung - ultrastructure Male Malondialdehyde - metabolism Microscopy, Electron, Transmission Organometallic Compounds - toxicity Oxidative Stress Protein Kinases - genetics Protein Kinases - metabolism Rats Rats, Sprague-Dawley RNA, Messenger - metabolism 一氧化碳 动物实验 大鼠 急性 损伤机制 羰基镍 肺炎 金属化合物 |
| title | The Mechanism of Acute Lung Injury Induced by Nickel Carbonyl in Rats |
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