Acidosis in models of cardiac ventricular myocytes

Acidosis in models of cardiac ventricular myocytes

transients during acidosis. These interactions are incorporated into a dynamical model of pH regulation in the myocyte to simulate respiratory acidosis in the heart.

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Veröffentlicht in:Philosophical transactions of the Royal Society of London. Series A: Mathematical, physical, and engineering sciences physical, and engineering sciences, 2006-05, Vol.364 (1842), p.1171-1186
Hauptverfasser: Crampin, Edmund J, Smith, Nicolas P, Langham, A. Elise, Clayton, Richard H, Orchard, Clive H
Format: Artikel
Sprache:eng
Schlagworte:
Acidosis
Acidosis - physiopathology
Action Potentials
Animals
Calcium Signaling
Cardiac Ventricular Myocyte
Cell membranes
Cells, Cultured
Computer Simulation
Heart Ventricles - chemistry
Heart Ventricles - cytology
Heart Ventricles - physiopathology
Humans
Hydrogen-Ion Concentration
Intracellular Calcium Handling
Mathematical Model
Membrane Potentials
Modeling
Models, Cardiovascular
Muscle Cells
Myocardium
Orchards
Ph Regulation
Physiological regulation
Protons
Respiratory acidosis
Ventricular Dysfunction, Left - physiopathology
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Beschreibung
Zusammenfassung:transients during acidosis. These interactions are incorporated into a dynamical model of pH regulation in the myocyte to simulate respiratory acidosis in the heart.
ISSN:1364-503X
1471-2962
DOI:10.1098/rsta.2006.1763