Cyclophosphamide Decreases Nitrotyrosine Formation and Inhibits Nitric Oxide Production by Alveolar Macrophages in Mycoplasmosis

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Veröffentlicht in:	Infection and Immunity 2001-10, Vol.69 (10), p.6401-6410
Hauptverfasser:	HICKMAN-DAVIS, Judy M, LINDSEY, J. Russell, MATALON, Sadis
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Bacteriology Biological and medical sciences Cells, Cultured cyclophosphamide Cyclophosphamide - administration & dosage Cyclophosphamide - therapeutic use Disease Models, Animal Fundamental and applied biological sciences. Psychology Host Response and Inflammation Immunosuppressive Agents - administration & dosage Immunosuppressive Agents - therapeutic use Leukocyte Count Life cycle. Host-agent relationship. Pathogenesis Lung - pathology Macrophages, Alveolar - cytology Macrophages, Alveolar - drug effects Macrophages, Alveolar - immunology Mice Mice, Inbred C57BL Mice, Knockout Microbiology Mycoplasma - drug effects Mycoplasma Infections - drug therapy Mycoplasma Infections - immunology Mycoplasma Infections - microbiology Mycoplasma Infections - pathology Mycoplasma pulmonis myeloperoxidase Neutrophils - cytology nitric oxide Nitric Oxide - biosynthesis nitrotyrosine Pathogenicity, virulence, toxins, bacteriocins, pyrogens, host-bacteria relations, miscellaneous strains Protozoa Tyrosine - analogs & derivatives Tyrosine - biosynthesis
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Zusammenfassung:	Classifications Services IAI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit StumbleUpon Twitter current issue Spotlights in the Current Issue IAI About IAI Subscribers Authors Reviewers Advertisers Inquiries from the Press Permissions & Commercial Reprints ASM Journals Public Access Policy Connect to IAI IAI RSS Feeds 1752 N Street N.W. • Washington DC 20036 202.737.3600 • 202.942.9355 fax • journals@asmusa.org Print ISSN: 0019-9567 Online ISSN: 1098-5522 Copyright © 2014 by the American Society for Microbiology. For an alternate route to IAI .asm.org, visit: IAI
ISSN:	0019-9567 1098-5522
DOI:	10.1128/IAI.69.10.6401-6410.2001