Mechanisms of Congenital Heart Disease Caused by NAA15 Haploinsufficiency

[Figure: see text].

Gespeichert in:

Bibliographische Detailangaben
Veröffentlicht in:	Circulation research 2021-04, Vol.128 (8), p.1156-1169
Hauptverfasser:	Ward, Tarsha, Tai, Warren, Morton, Sarah, Impens, Francis, Van Damme, Petra, Van Haver, Delphi, Timmerman, Evy, Venturini, Gabriela, Zhang, Kehan, Jang, Min Young, Willcox, Jon A.L., Haghighi, Alireza, Gelb, Bruce D., Chung, Wendy K., Goldmuntz, Elizabeth, Porter, George A., Lifton, Richard P., Brueckner, Martina, Yost, H. Joseph, Bruneau, Benoit G., Gorham, Joshua, Kim, Yuri, Pereira, Alexandre, Homsy, Jason, Benson, Craig C., DePalma, Steven R., Varland, Sylvia, Chen, Christopher S., Arnesen, Thomas, Gevaert, Kris, Seidman, Christine, Seidman, J.G.
Format:	Artikel
Sprache:	eng
Schlagworte:	Acetylation Cells, Cultured Child Haploinsufficiency Heart Defects, Congenital - genetics Heart Defects, Congenital - metabolism Heart Defects, Congenital - pathology Humans Induced Pluripotent Stem Cells - metabolism Mutation, Missense N-Terminal Acetyltransferase A - genetics N-Terminal Acetyltransferase E - genetics Original Research Protein Processing, Post-Translational Proteome - metabolism
Online-Zugang:	Volltext
Tags:	Tag hinzufügen Keine Tags, Fügen Sie den ersten Tag hinzu!