Lack of Gdf11 does not improve anemia or prevent the activity of RAP-536 in a mouse model of β-thalassemia

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Bibliographische Detailangaben
Veröffentlicht in:	Blood 2019-08, Vol.134 (6), p.568-572
Hauptverfasser:	Guerra, Amaliris, Oikonomidou, Paraskevi Rea, Sinha, Sayantani, Zhang, Jianbing, Lo Presti, Vania, Hamilton, Callum R., Breda, Laura, Casu, Carla, La, Ping, Martins, Ana C., Sendamarai, Anoop K., Fleming, Mark, Rivella, Stefano
Format:	Artikel
Sprache:	eng
Schlagworte:	Anemia - blood Anemia - drug therapy Anemia - etiology Animals beta-Thalassemia - blood beta-Thalassemia - drug therapy beta-Thalassemia - genetics beta-Thalassemia - metabolism Biomarkers Bone Morphogenetic Proteins - deficiency Bone Morphogenetic Proteins - genetics Bone Morphogenetic Proteins - metabolism Disease Models, Animal Growth Differentiation Factors - deficiency Growth Differentiation Factors - genetics Growth Differentiation Factors - metabolism Letter to Mice Mice, Transgenic Recombinant Fusion Proteins - pharmacology Treatment Outcome
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Beschreibung
Zusammenfassung:	There is a Blood Commentary on this article in this issue.
ISSN:	0006-4971 1528-0020
DOI:	10.1182/blood.2019001057