Inactivation of fatty acid synthase impairs hepatocarcinogenesis driven by AKT in mice and humans

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Veröffentlicht in:	Journal of hepatology 2016-02, Vol.64 (2), p.333-341
Hauptverfasser:	Li, Lei, Pilo, Giulia M, Li, Xiaolei, Cigliano, Antonio, Latte, Gavinella, Che, Li, Joseph, Christy, Mela, Marta, Wang, Chunmei, Jiang, Lijie, Ribback, Silvia, Simile, Maria M, Pascale, Rosa M, Dombrowski, Frank, Evert, Matthias, Semenkovich, Clay F, Chen, Xin, Calvisi, Diego F
Format:	Artikel
Sprache:	eng
Schlagworte:	AKT Animals Apoptosis - physiology Carcinogenesis - genetics Carcinogenesis - metabolism Carcinoma, Hepatocellular - genetics Carcinoma, Hepatocellular - pathology Cell Line, Tumor Cell Proliferation - physiology Fatty acid synthase Fatty Acid Synthase, Type I - genetics Fatty Acid Synthase, Type I - metabolism Gastroenterology and Hepatology Gene Expression Regulation, Neoplastic - physiology Hepatocellular carcinoma Humans Lipogenesis Liver - metabolism Liver - pathology Liver Neoplasms - genetics Liver Neoplasms - pathology Mice Phosphorylation Proto-Oncogene Proteins c-akt - metabolism Rictor Signal Transduction - genetics
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