The ubiquitin editing enzyme A20 (TNFAIP3) is upregulated during permanent middle cerebral artery occlusion but does not influence disease outcome

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Bibliographische Detailangaben
Veröffentlicht in:	Cell death & disease 2013-03, Vol.4 (3), p.e531-e531
Hauptverfasser:	Mc Guire, C, Rahman, M, Schwaninger, M, Beyaert, R, van Loo, G
Format:	Artikel
Sprache:	eng
Schlagworte:	631/80/82 692/699/375/1370 Animals Antibodies Biochemistry Biomedical and Life Sciences Cell Biology Cell Culture Correspondence Cysteine Endopeptidases - deficiency Cysteine Endopeptidases - genetics Cysteine Endopeptidases - metabolism Immunology Infarction, Middle Cerebral Artery - metabolism Infarction, Middle Cerebral Artery - pathology Interleukin-6 - metabolism Intracellular Signaling Peptides and Proteins - deficiency Intracellular Signaling Peptides and Proteins - genetics Intracellular Signaling Peptides and Proteins - metabolism Life Sciences Mice Mice, Inbred C57BL NF-kappa B - metabolism Signal Transduction Tumor Necrosis Factor alpha-Induced Protein 3 Tumor Necrosis Factors - metabolism Up-Regulation
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