Inhibition of glycogen synthase kinase-3 is necessary for the rapid antidepressant effect of ketamine in mice

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Veröffentlicht in:	Molecular psychiatry 2011-11, Vol.16 (11), p.1068-1070
Hauptverfasser:	Beurel, E, Song, L, Jope, R S
Format:	Artikel
Sprache:	eng
Schlagworte:	631/92/436/2388 692/699/476/1414 Amino Acid Substitution Animals Antidepressants Antidepressive Agents - pharmacology Behavioral Sciences Biological and medical sciences Biological Psychology Cerebral Cortex - drug effects Cerebral Cortex - enzymology Depression - drug therapy Depression, Mental Disease Models, Animal Drug Evaluation, Preclinical Drug therapy Electroshock Gene Knock-In Techniques Glycogen Glycogen synthase kinase 3 Glycogen Synthase Kinase 3 - antagonists & inhibitors Glycogen Synthase Kinase 3 - chemistry Glycogen Synthase Kinase 3 - genetics Glycogen Synthase Kinase 3 - physiology Glycogen Synthase Kinase 3 beta Health aspects Helplessness, Learned Hippocampus - drug effects Hippocampus - enzymology Ketamine Ketamine - pharmacology letter-to-the-editor Lithium Chloride - pharmacology Medical sciences Medicine Medicine & Public Health Mice Nerve Tissue Proteins - antagonists & inhibitors Nerve Tissue Proteins - chemistry Nerve Tissue Proteins - genetics Nerve Tissue Proteins - physiology Neuropharmacology Neurosciences Pharmacology. Drug treatments Pharmacotherapy Phosphorylation - drug effects Phosphoserine - metabolism Phosphotransferases Physiological aspects Point Mutation Protein Processing, Post-Translational - drug effects Psychiatry Psychoanaleptics: cns stimulant, antidepressant agent, nootropic agent, mood stabilizer Psychoanaleptics: cns stimulant, antidepressant agent, nootropic agent, mood stabilizer..., (alzheimer disease) Psychology. Psychoanalysis. Psychiatry Psychopathology. Psychiatry Psychopharmacology
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ISSN:	1359-4184 1476-5578
DOI:	10.1038/mp.2011.47