Altered sodium channel function in dystrophin/utrophin-deficient cardiomyocytes

Meeting abstracts - A single PDF containing all abstracts in this Supplement is available at http://www.biomedcentral.com/content/files/pdf/1471-2210-10-S1-full.pdf.EMPTY Background Duchenne muscular dystrophy (DMD), caused by mutations in the dystrophin gene, is an inherited disease characterized by progressive muscle weakness and degeneration. Besides the relatively well-described skeletal muscle degenerative processes, DMD and some other muscular dystrophy types are also associated with cardiovascular complications including cardiomyopathy and cardiac arrhythmias.