Repression of IP-10 by Interactions between Histone Deacetylation and Hypermethylation in Idiopathic Pulmonary Fibrosis

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Veröffentlicht in:	Molecular and Cellular Biology 2010-06, Vol.30 (12), p.2874-2886
Hauptverfasser:	Coward, William R., Watts, Keira, Feghali-Bostwick, Carol A., Jenkins, Gisli, Pang, Linhua
Format:	Artikel
Sprache:	eng
Schlagworte:	Acetylation - drug effects Cell Line Chemokine CXCL10 - genetics Chemokine CXCL10 - metabolism Chromosomal Proteins, Non-Histone - metabolism Cytokines - pharmacology Histone Acetyltransferases - metabolism Histone Deacetylase Inhibitors - pharmacology Histone Deacetylases - metabolism Histone-Lysine N-Methyltransferase - antagonists & inhibitors Histone-Lysine N-Methyltransferase - metabolism Histones - metabolism Humans Idiopathic Pulmonary Fibrosis - genetics Idiopathic Pulmonary Fibrosis - metabolism Methylation - drug effects Promoter Regions, Genetic - genetics Protein Binding - drug effects Repressor Proteins - metabolism RNA, Messenger - genetics RNA, Messenger - metabolism Transcription Factors - metabolism
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ISSN:	0270-7306 1098-5549 1098-5549
DOI:	10.1128/MCB.01527-09