The t(12;21) translocation converts AML-1B from an activator to a repressor of transcription

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Veröffentlicht in:	Molecular and Cellular Biology 1996-04, Vol.16 (4), p.1349-1355
Hauptverfasser:	Hiebert, Scott W., Sun, Wuhua, Nathan Davis, J., Golub, Todd, Shurtleff, Sheila, Buijs, Arjan, Downing, James R., Grosveld, Gerard, Roussel, Martine F., Gary Gilliland, D., Lenny, Noel, Meyers, Shari
Format:	Artikel
Sprache:	eng
Schlagworte:	Base Sequence Chromosomes, Human, Pair 12 Chromosomes, Human, Pair 21 DNA-Binding Proteins - genetics Enhancer Elements, Genetic - genetics ETS Translocation Variant 6 Protein Helix-Loop-Helix Motifs Humans Leukemia - genetics Molecular Sequence Data Proto-Oncogene Proteins c-ets Recombinant Fusion Proteins - genetics Repressor Proteins Sequence Deletion Transcription Factors - genetics Transcription, Genetic Translocation, Genetic
Online-Zugang:	Volltext
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Zusammenfassung:	Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit StumbleUpon Twitter current issue Spotlights in the Current Issue MCB About MCB Subscribers Authors Reviewers Advertisers Inquiries from the Press Permissions & Commercial Reprints ASM Journals Public Access Policy MCB RSS Feeds 1752 N Street N.W. • Washington DC 20036 202.737.3600 • 202.942.9355 fax • journals@asmusa.org Print ISSN: 0270-7306 Online ISSN: 1098-5549 Copyright © 2014 by the American Society for Microbiology. For an alternate route to MCB .asm.org, visit: MCB
ISSN:	0270-7306 1098-5549 1098-5549
DOI:	10.1128/MCB.16.4.1349