Disease progression and liver cancer in the ferroportin disease
In 2006, at the age of 83 years, hepatic ultrasound (US) examination of the proband identified two hyperechoic nodules in the liver; contrast-enhanced MRI scan confirmed the US examination findings and a US-guided liver biopsy documented a well-differentiated hepatocellular carcinoma (fig 1A), as sh...
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Veröffentlicht in: | Gut 2007-07, Vol.56 (7), p.1030-1032 |
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creator | Corradini, Elena Ferrara, Francesca Pollicino, Teresa Vegetti, Alberto Abbati, Gian Luca Losi, Luisa Raimondo, Giovanni Pietrangelo, Antonello |
description | In 2006, at the age of 83 years, hepatic ultrasound (US) examination of the proband identified two hyperechoic nodules in the liver; contrast-enhanced MRI scan confirmed the US examination findings and a US-guided liver biopsy documented a well-differentiated hepatocellular carcinoma (fig 1A), as shown by antihepatocyte antibody stain (not shown here), in the absence of cirrhosis. [...]the ferroportin disease may progress if not properly treated, and complicate with liver cancer even without cirrhosis, in the presence of cocarcinogenic factors, such as occult HBV infection. |
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[...]the ferroportin disease may progress if not properly treated, and complicate with liver cancer even without cirrhosis, in the presence of cocarcinogenic factors, such as occult HBV infection.</description><identifier>ISSN: 0017-5749</identifier><identifier>EISSN: 1468-3288</identifier><identifier>DOI: 10.1136/gut.2007.122549</identifier><identifier>PMID: 17566043</identifier><identifier>CODEN: GUTTAK</identifier><language>eng</language><publisher>England: BMJ Publishing Group Ltd and British Society of Gastroenterology</publisher><subject>Age ; Biopsy ; Carcinoma, Hepatocellular - etiology ; Cation Transport Proteins - genetics ; Disease Progression ; Hepatitis ; Humans ; Iron ; Iron Overload - complications ; Letters ; Liver cancer ; Liver cirrhosis ; Liver Neoplasms - etiology ; Male ; Middle Aged ; Mutation ; Patients</subject><ispartof>Gut, 2007-07, Vol.56 (7), p.1030-1032</ispartof><rights>Copyright 2007 by Gut</rights><rights>Copyright: 2007 Copyright 2007 by Gut</rights><rights>Copyright © 2007 BMJ Publishing Group & British Society of Gastroenterology</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-b527t-d82902e91811d5f52de5c8c05529e150c00e887b97b5d7f7c996edf19dd55c403</citedby></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttp://gut.bmj.com/content/56/7/1030.full.pdf$$EPDF$$P50$$Gbmj$$H</linktopdf><linktohtml>$$Uhttp://gut.bmj.com/content/56/7/1030.full$$EHTML$$P50$$Gbmj$$H</linktohtml><link.rule.ids>114,115,230,314,723,776,780,881,3183,23550,27901,27902,53766,53768,77342,77373</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/17566043$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Corradini, Elena</creatorcontrib><creatorcontrib>Ferrara, Francesca</creatorcontrib><creatorcontrib>Pollicino, Teresa</creatorcontrib><creatorcontrib>Vegetti, Alberto</creatorcontrib><creatorcontrib>Abbati, Gian Luca</creatorcontrib><creatorcontrib>Losi, Luisa</creatorcontrib><creatorcontrib>Raimondo, Giovanni</creatorcontrib><creatorcontrib>Pietrangelo, Antonello</creatorcontrib><title>Disease progression and liver cancer in the ferroportin disease</title><title>Gut</title><addtitle>Gut</addtitle><description>In 2006, at the age of 83 years, hepatic ultrasound (US) examination of the proband identified two hyperechoic nodules in the liver; contrast-enhanced MRI scan confirmed the US examination findings and a US-guided liver biopsy documented a well-differentiated hepatocellular carcinoma (fig 1A), as shown by antihepatocyte antibody stain (not shown here), in the absence of cirrhosis. [...]the ferroportin disease may progress if not properly treated, and complicate with liver cancer even without cirrhosis, in the presence of cocarcinogenic factors, such as occult HBV infection.</description><subject>Age</subject><subject>Biopsy</subject><subject>Carcinoma, Hepatocellular - etiology</subject><subject>Cation Transport Proteins - genetics</subject><subject>Disease Progression</subject><subject>Hepatitis</subject><subject>Humans</subject><subject>Iron</subject><subject>Iron Overload - complications</subject><subject>Letters</subject><subject>Liver cancer</subject><subject>Liver cirrhosis</subject><subject>Liver Neoplasms - etiology</subject><subject>Male</subject><subject>Middle Aged</subject><subject>Mutation</subject><subject>Patients</subject><issn>0017-5749</issn><issn>1468-3288</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2007</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><sourceid>BENPR</sourceid><recordid>eNqFkMtLAzEQh4MoWh9nb7LgTdiax2aTXBRp1QpFL7XXsJvM1q3tpibbov-9kS0-Tp6GYb75zfAhdEpwnxCWX87WbZ9iLPqEUp6pHdQjWS5TRqXcRT2MiUi5yNQBOgxhjjGWUpF9dEAEz3OcsR66HtYBigDJyruZhxBq1yRFY5NFvQGfmKIxsdRN0r5AUoH3buV8G3vb7R2jvapYBDjZ1iP0fHc7GYzS8dP9w-BmnJacija1kipMQRFJiOUVpxa4kQZzThUQjg3GIKUolSi5FZUwSuVgK6Ks5dxkmB2hqy53tS6XYA00rS8WeuXrZeE_tCtq_XfS1C965jaaKJUxIWLA-TbAu7c1hFbP3do38WdNhFCMSZmzSF12lPEuBA_V9wWC9ZdxHY3rL-O6Mx43zn4_9sNvFUcg7YA6tPD-PS_8q84FE1w_Tgf6fjocjkcjoSeRv-j4cjn_9_onV_KZTA</recordid><startdate>200707</startdate><enddate>200707</enddate><creator>Corradini, Elena</creator><creator>Ferrara, Francesca</creator><creator>Pollicino, Teresa</creator><creator>Vegetti, Alberto</creator><creator>Abbati, Gian Luca</creator><creator>Losi, Luisa</creator><creator>Raimondo, Giovanni</creator><creator>Pietrangelo, Antonello</creator><general>BMJ Publishing Group Ltd and British Society of Gastroenterology</general><general>BMJ Publishing Group LTD</general><general>BMJ Group</general><scope>BSCLL</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>3V.</scope><scope>7X7</scope><scope>7XB</scope><scope>88E</scope><scope>88I</scope><scope>8AF</scope><scope>8FE</scope><scope>8FH</scope><scope>8FI</scope><scope>8FJ</scope><scope>8FK</scope><scope>ABUWG</scope><scope>AFKRA</scope><scope>AZQEC</scope><scope>BBNVY</scope><scope>BENPR</scope><scope>BHPHI</scope><scope>BTHHO</scope><scope>CCPQU</scope><scope>DWQXO</scope><scope>FYUFA</scope><scope>GHDGH</scope><scope>GNUQQ</scope><scope>HCIFZ</scope><scope>K9.</scope><scope>LK8</scope><scope>M0S</scope><scope>M1P</scope><scope>M2P</scope><scope>M7P</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>PRINS</scope><scope>Q9U</scope><scope>5PM</scope></search><sort><creationdate>200707</creationdate><title>Disease progression and liver cancer in the ferroportin disease</title><author>Corradini, Elena ; 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contrast-enhanced MRI scan confirmed the US examination findings and a US-guided liver biopsy documented a well-differentiated hepatocellular carcinoma (fig 1A), as shown by antihepatocyte antibody stain (not shown here), in the absence of cirrhosis. 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subjects | Age Biopsy Carcinoma, Hepatocellular - etiology Cation Transport Proteins - genetics Disease Progression Hepatitis Humans Iron Iron Overload - complications Letters Liver cancer Liver cirrhosis Liver Neoplasms - etiology Male Middle Aged Mutation Patients |
title | Disease progression and liver cancer in the ferroportin disease |
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