Discussion: Immunology, cellular pathology, genetics
[...]recent studies from Zambia indicate that PsA improves with the onset of AIDS in human immunodeficiency virus (HIV) positive patients. [...]a dominant role for T cells in PsA has been challenged by a recent randomised study showing that efalizumab, an agent that inhibits T cells, significantly l...
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Veröffentlicht in: | Annals of the rheumatic diseases 2005-03, Vol.64 (suppl 2), p.ii40-ii41 |
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container_title | Annals of the rheumatic diseases |
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creator | Ritchlin, C |
description | [...]recent studies from Zambia indicate that PsA improves with the onset of AIDS in human immunodeficiency virus (HIV) positive patients. [...]a dominant role for T cells in PsA has been challenged by a recent randomised study showing that efalizumab, an agent that inhibits T cells, significantly lessened psoriasis activity but not joint inflammation in patients with PsA. |
doi_str_mv | 10.1136/ard.2004.031559 |
format | Article |
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[...]a dominant role for T cells in PsA has been challenged by a recent randomised study showing that efalizumab, an agent that inhibits T cells, significantly lessened psoriasis activity but not joint inflammation in patients with PsA.</description><identifier>ISSN: 0003-4967</identifier><identifier>EISSN: 1468-2060</identifier><identifier>DOI: 10.1136/ard.2004.031559</identifier><identifier>CODEN: ARDIAO</identifier><language>eng</language><publisher>Kidlington: BMJ Publishing Group Ltd and European League Against Rheumatism</publisher><subject>Antigens ; Arthritis ; cellular pathology ; Cytokines ; dendritic cell ; Discussion ; Genetics ; HIV ; HLA ; Human immunodeficiency virus ; human leucocyte antigen ; Immunology ; Inflammation ; interleukin ; Lymphocytes ; major histocompatibility complex ; Membranes ; MHC ; Morphology ; Pathogenesis ; Pathology ; Population ; PsA ; Psoriasis ; psoriatic arthritis ; psoriatic arthritis genetics ; RANKL ; receptor activator of nuclear factor κB ligand ; rheumatoid arthritis ; Studies ; TNF ; Tumor necrosis factor-TNF ; tumour necrosis factor</subject><ispartof>Annals of the rheumatic diseases, 2005-03, Vol.64 (suppl 2), p.ii40-ii41</ispartof><rights>Copyright 2005 by Annals of the Rheumatic Diseases</rights><rights>Copyright: 2005 Copyright 2005 by Annals of the Rheumatic Diseases</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC1766873/pdf/$$EPDF$$P50$$Gpubmedcentral$$H</linktopdf><linktohtml>$$Uhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC1766873/$$EHTML$$P50$$Gpubmedcentral$$H</linktohtml><link.rule.ids>230,314,723,776,780,881,27901,27902,53766,53768</link.rule.ids></links><search><creatorcontrib>Ritchlin, C</creatorcontrib><title>Discussion: Immunology, cellular pathology, genetics</title><title>Annals of the rheumatic diseases</title><addtitle>Ann Rheum Dis</addtitle><description>[...]recent studies from Zambia indicate that PsA improves with the onset of AIDS in human immunodeficiency virus (HIV) positive patients. [...]a dominant role for T cells in PsA has been challenged by a recent randomised study showing that efalizumab, an agent that inhibits T cells, significantly lessened psoriasis activity but not joint inflammation in patients with PsA.</description><subject>Antigens</subject><subject>Arthritis</subject><subject>cellular pathology</subject><subject>Cytokines</subject><subject>dendritic cell</subject><subject>Discussion</subject><subject>Genetics</subject><subject>HIV</subject><subject>HLA</subject><subject>Human immunodeficiency virus</subject><subject>human leucocyte antigen</subject><subject>Immunology</subject><subject>Inflammation</subject><subject>interleukin</subject><subject>Lymphocytes</subject><subject>major histocompatibility complex</subject><subject>Membranes</subject><subject>MHC</subject><subject>Morphology</subject><subject>Pathogenesis</subject><subject>Pathology</subject><subject>Population</subject><subject>PsA</subject><subject>Psoriasis</subject><subject>psoriatic arthritis</subject><subject>psoriatic arthritis genetics</subject><subject>RANKL</subject><subject>receptor activator of nuclear factor κB ligand</subject><subject>rheumatoid arthritis</subject><subject>Studies</subject><subject>TNF</subject><subject>Tumor necrosis factor-TNF</subject><subject>tumour necrosis 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subjects | Antigens Arthritis cellular pathology Cytokines dendritic cell Discussion Genetics HIV HLA Human immunodeficiency virus human leucocyte antigen Immunology Inflammation interleukin Lymphocytes major histocompatibility complex Membranes MHC Morphology Pathogenesis Pathology Population PsA Psoriasis psoriatic arthritis psoriatic arthritis genetics RANKL receptor activator of nuclear factor κB ligand rheumatoid arthritis Studies TNF Tumor necrosis factor-TNF tumour necrosis factor |
title | Discussion: Immunology, cellular pathology, genetics |
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