Hypopituitarism following cerebral oedema with diabetic ketoacidosis

Clinical evidence of cerebral oedema occurs in approximately 1% of diabetic ketoacidosis episodes. Mortality from this serious complication is falling, but little is known of long term outcome. We describe hypopituitarism and executive dysfunction developing two years after cerebral oedema complicat...

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Veröffentlicht in:	Archives of disease in childhood 2002-10, Vol.87 (4), p.337-338
Hauptverfasser:	Dunlop, K A, Woodman, D, Carson, D J
Format:	Artikel
Sprache:	eng
Schlagworte:	Age Associated diseases and complications Biological and medical sciences Brain Brain Edema - etiology Case Report Cerebral edema cerebral oedema Child Complications and side effects computed tomography Consciousness Diabetes Diabetes Mellitus, Type 1 - complications Diabetes. Impaired glucose tolerance Diabetic acidosis Diabetic ketoacidosis Diabetic Ketoacidosis - complications DKA Endocrine pancreas. Apud cells (diseases) Endocrine therapy Endocrinopathies Follow-Up Studies free thyroxine FT4 Growth Disorders - etiology Growth hormones height velocity Htvel Humans Hypopituitarism Hypopituitarism - etiology IGF Insulin insulin-like growth factor Insulin-like growth factors Intensive care Ketoacidosis Learning Problems Male Medical imaging Medical sciences Motion Ostomy Pituitary gland Psychometrics Stimuli thyroid stimulating hormone TSH Velocity Visual Impairments
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Zusammenfassung:	Clinical evidence of cerebral oedema occurs in approximately 1% of diabetic ketoacidosis episodes. Mortality from this serious complication is falling, but little is known of long term outcome. We describe hypopituitarism and executive dysfunction developing two years after cerebral oedema complicating diabetic ketoacidosis in a 12 year old with type 1 diabetes.
ISSN:	0003-9888 1468-2044
DOI:	10.1136/adc.87.4.337