Human GLI-2 Is a Tat Activation Response Element-Independent Tat Cofactor

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Veröffentlicht in:	Journal of Virology 2001-03, Vol.75 (5), p.2314-2323
Hauptverfasser:	Browning, C M, Smith, M J, Clark, N M, Lane, B R, Parada, C, Montano, M, KewalRamani, V N, Littman, D R, Essex, M, Roeder, R G, Markovitz, D M
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Bacterial Proteins - metabolism Cell Line Chemoreceptor Cells Cyclin T cyclin T1 Cyclins - metabolism Drosophila Proteins Escherichia coli Proteins Gene Products, tat - genetics Gene Products, tat - metabolism GLI-2 protein HIV Infections - virology HIV-1 - genetics HIV-1 - physiology HIV-2 - genetics HIV-2 - physiology Human immunodeficiency virus 1 Human immunodeficiency virus 2 Humans Kruppel-Like Transcription Factors Membrane Proteins - metabolism Mice Nuclear Proteins Plasmids Receptors, Cell Surface Response Elements - genetics tat Gene Products, Human Immunodeficiency Virus TATA Box - physiology Transcription Factors - genetics Transcription Factors - metabolism Transcriptional Activation Transfection Virus Replication Virus-Cell Interactions Zinc Finger Protein Gli2
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Zusammenfassung:	Article Usage Stats Services JVI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit StumbleUpon Twitter current issue Spotlights in the Current Issue JVI About JVI Subscribers Authors Reviewers Advertisers Inquiries from the Press Permissions & Commercial Reprints ASM Journals Public Access Policy JVI RSS Feeds 1752 N Street N.W. • Washington DC 20036 202.737.3600 • 202.942.9355 fax • journals@asmusa.org Print ISSN: 0022-538X Online ISSN: 1098-5514 Copyright © 2014 by the American Society for Microbiology. For an alternate route to JVI .asm.org, visit: JVI
ISSN:	0022-538X 1098-5514
DOI:	10.1128/JVI.75.5.2314-2323.2001