Occupancy of whole blood cells by a single PIGA‐mutant clone with HMGA2 amplification in a paroxysmal nocturnal haemoglobinuria patient having blood cells with NKG2D ligands

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Veröffentlicht in:	British journal of haematology 2013-01, Vol.160 (1), p.114-116
Hauptverfasser:	Hanaoka, Nobuyoshi, Murakami, Yoshiko, Nagata, Masahide, Horikawa, Kentaro, Nagakura, Shoichi, Yonemura, Yuji, Murata, Shogo, Sonoki, Takashi, Kinoshita, Taroh, Nakakuma, Hideki
Format:	Artikel
Sprache:	eng
Schlagworte:	Aged Anemias. Hemoglobinopathies Biological and medical sciences clonal expansion Diseases of red blood cells Female Gene Expression Regulation Hematologic and hematopoietic diseases Hemoglobinuria, Paroxysmal - blood Hemoglobinuria, Paroxysmal - genetics Hemoglobinuria, Paroxysmal - immunology high mobility group AT‐hook 2 HMGA2 Protein - biosynthesis HMGA2 Protein - blood HMGA2 Protein - genetics Humans immunoselection Ligands Medical sciences Membrane Proteins - blood Membrane Proteins - genetics NK Cell Lectin-Like Receptor Subfamily K - blood NK Cell Lectin-Like Receptor Subfamily K - immunology NKG2D (KLRK1) paroxysmal nocturnal haemoglobinuria Tumors
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