Anti-TNFα therapy may lead to blood pressure reductions through improved endothelium-dependent microvascular function in patients with rheumatoid arthritis

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Veröffentlicht in:	Journal of human hypertension 2011-11, Vol.25 (11), p.699-702
Hauptverfasser:	Sandoo, A, Panoulas, V F, Toms, T E, Smith, J P, Stavropoulos-Kalinoglou, A, Metsios, G S, Gasparyan, A Y, Carroll, D, Veldhuijzen van Zanten, J J C S, Kitas, G D
Format:	Artikel
Sprache:	eng
Schlagworte:	631/443/1338/243 692/420 692/699/249/1313/498 692/700/565/251 Adalimumab Antibodies, Monoclonal - therapeutic use Antibodies, Monoclonal, Humanized - therapeutic use Antirheumatic Agents - therapeutic use Arterial hypertension. Arterial hypotension Arthritis, Rheumatoid - drug therapy Arthritis, Rheumatoid - immunology Arthritis, Rheumatoid - physiopathology Biological and medical sciences Blood and lymphatic vessels Blood pressure Blood Pressure - drug effects Cardiology. Vascular system Clinical manifestations. Epidemiology. Investigative techniques. Etiology Endothelium Endothelium, Vascular - drug effects Endothelium, Vascular - immunology Endothelium, Vascular - physiopathology England Epidemiology Etanercept Health Administration Humans Immunoglobulin G - therapeutic use Inflammation Mediators - blood Infliximab Longitudinal Studies Medical sciences Medicine Medicine & Public Health Microvasculature Microvessels - drug effects Microvessels - immunology Microvessels - physiopathology Public Health Receptors, Tumor Necrosis Factor - therapeutic use research-letter Rheumatoid arthritis Time Factors Treatment Outcome Tumor Necrosis Factor-alpha - antagonists & inhibitors Tumor Necrosis Factor-alpha - blood Tumor necrosis factor-α
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ISSN:	0950-9240 1476-5527
DOI:	10.1038/jhh.2011.36