IgA Nephropathy Associated with Portal Hypertension in Liver Cirrhosis due to Non-Alcoholic and Non-A, Non-B, Non-C Hepatitis

A 69-year-old female was admitted to our hospital because of leg edema, proteinuria (2.1 g/day), and gross hematuria. She had non-alcoholic liver cirrhosis of unknown etiology. Esophageal varices also were found. Examination of the renal biopsy specimen revealed mesangial proliferative glomeruloneph...

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Veröffentlicht in:	Internal Medicine 1994, Vol.33(8), pp.488-491
Hauptverfasser:	NAKAMURA, Mono, OHISHI, Akira, WATANABE, Reiko, KANEKO, Kohtaro, AOSAKI, Noboru, IIGAYA, Tomohiko, MONMA, Tetsuo, SUGIURA, Hitoshi, MIYOSHI, Yukari, HAMAGUCHI, Kinichi
Format:	Artikel
Sprache:	eng
Schlagworte:	Aged Female Glomerulonephritis, IGA - etiology Glomerulonephritis, IGA - pathology Hepatitis - complications Hepatitis - pathology Humans Hypertension, Portal - drug therapy Hypertension, Portal - etiology Hypertension, Portal - pathology Kidney Glomerulus - ultrastructure Liver Cirrhosis - etiology Liver Cirrhosis - pathology liver disease mesangial proliferative glomerulonephritis Microscopy, Electron Propranolol - therapeutic use β-blocker
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Zusammenfassung:	A 69-year-old female was admitted to our hospital because of leg edema, proteinuria (2.1 g/day), and gross hematuria. She had non-alcoholic liver cirrhosis of unknown etiology. Esophageal varices also were found. Examination of the renal biopsy specimen revealed mesangial proliferative glomerulonephritis with IgA deposits. Propranolol was administered orally to reduce portal hypertension, resulting in a progressive decrease in urinary microalbumin excretion. This case suggests that portal hypertension is involved in the pathogenesis of IgA nephropathy in liver cirrhosis. (Internal Medicine 33: 488-491, 1994)
ISSN:	0918-2918 1349-7235
DOI:	10.2169/internalmedicine.33.488