Interrelationships between sex and ractopamine on protein and lipid deposition in rapidly growing pigs

Sixty pigs were used to investigate the effects of two levels of dietary ractopamine (RAC; 0 and 20 mg/kg) and three sex types (SEX; boars, gilts, and barrows) on performance over the live weight range 60 to 90 kg. Pigs were housed in individual pens and allowed ad libitum access to a diet containin...

Ausführliche Beschreibung

Gespeichert in:
Bibliographische Detailangaben
Veröffentlicht in:Journal of animal science 1993-11, Vol.71 (11), p.2919-2930
Hauptverfasser: Dunshea, F.R, King, R.H, Campbell, R.G, Sainz, R.D, Kim, Y.S
Format: Artikel
Sprache:eng
Schlagworte:
Online-Zugang:Volltext
Tags: Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
Beschreibung
Zusammenfassung:Sixty pigs were used to investigate the effects of two levels of dietary ractopamine (RAC; 0 and 20 mg/kg) and three sex types (SEX; boars, gilts, and barrows) on performance over the live weight range 60 to 90 kg. Pigs were housed in individual pens and allowed ad libitum access to a diet containing 3.466 Mcal of DE and 10.7 g of lysine/kg. Control boars exhibited faster and more efficient growth and deposited more protein and less fat than gilts or barrows. The RAC increased ADG by 17 and 21% in gilts and barrows but not in boars. Feed intake was not altered by dietary RAC. Dietary RAC increased the rate of protein deposition by 15, 42, and 41% in boars, gilts, and barrows, respectively. Nevertheless, the daily rate of protein deposition was greatest in RAC-treated boars. The RAC tended to reduce the daily rate of fat deposition by 21% in boars but not in gilts or barrows. Carcass protein content increased by 5% and fat content decreased by 8% in response to RAC. These improvements in carcass composition occurred without compromising meat quality. Results show that RAC is a potent stimulator of protein deposition in finishing pigs. However, increased protein deposition is not necessarily at the expense of fat deposition
ISSN:0021-8812
1525-3163
DOI:10.2527/1993.71112919x