Further evidence that substance P is a mediator of both neurogenic inflammation and pain: two phenomena inhibited either by postsynaptic blockade of NK1 receptors or by presynaptic action of opioid receptor agonists

Further evidence that substance P is a mediator of both neurogenic inflammation and pain: two phenomena inhibited either by postsynaptic blockade of NK1 receptors or by presynaptic action of opioid receptor agonists

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Veröffentlicht in:Regulatory peptides 1993-07, Vol.46 (1-2), p.424-425
Hauptverfasser: Moussaoui, S, Carruette, A, Garret, C
Format: Artikel
Sprache:eng
Schlagworte:
Analgesics - pharmacology
Animals
Anticonvulsants - pharmacology
Electric Stimulation
Formaldehyde
Indoles - pharmacology
Inflammation - physiopathology
Isoindoles
Male
Mice
Mice, Inbred Strains
Morphine - pharmacology
Pain - physiopathology
Peripheral Nerves - drug effects
Peripheral Nerves - physiology
Pyrroles - pharmacology
Rats
Rats, Sprague-Dawley
Substance P - antagonists & inhibitors
Substance P - pharmacology
Thiophenes - pharmacology
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ISSN:0167-0115