Is There an Effect of Amiodarone on the Defibrillation Threshold?
The interaction between amiodarone and the defibrillation threshold (DFT) is still controversial. We present a case with dilated cardiomyopathy and recurrent sustained monomorphic ventricular tachycardia who received an automatic implantable cardioverter defibrillator (AICD) while under long-term am...
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| Veröffentlicht in: | Japanese Heart Journal 1993, Vol.34(2), pp.221-226 |
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| container_end_page | 226 |
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| container_issue | 2 |
| container_start_page | 221 |
| container_title | Japanese Heart Journal |
| container_volume | 34 |
| creator | GÜLDAL, Muharrem KARAOGUZ, Remzi AKALIN, Hakki BAYAR, Mustafa AKYOL, Turhan |
| description | The interaction between amiodarone and the defibrillation threshold (DFT) is still controversial. We present a case with dilated cardiomyopathy and recurrent sustained monomorphic ventricular tachycardia who received an automatic implantable cardioverter defibrillator (AICD) while under long-term amiodarone treatment. AICD implantation was performed without thoracotomy. The transvenous lead was inserted via a left subclavian vein puncture and the patch was placed on the lateral chest wall, submuscularly. At the time of implantation a 35J shock was not successful in converting ventricular fibrillation to sinus rhythm, but a 40J rescue shock was successful. After discontinuation of amiodarone, DFT measurements were repeated. Sixteen days later DFT was still higher than 34J, but 71 days later it decreased to 20J. |
| doi_str_mv | 10.1536/ihj.34.221 |
| format | Article |
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We present a case with dilated cardiomyopathy and recurrent sustained monomorphic ventricular tachycardia who received an automatic implantable cardioverter defibrillator (AICD) while under long-term amiodarone treatment. AICD implantation was performed without thoracotomy. The transvenous lead was inserted via a left subclavian vein puncture and the patch was placed on the lateral chest wall, submuscularly. At the time of implantation a 35J shock was not successful in converting ventricular fibrillation to sinus rhythm, but a 40J rescue shock was successful. After discontinuation of amiodarone, DFT measurements were repeated. Sixteen days later DFT was still higher than 34J, but 71 days later it decreased to 20J.</description><identifier>ISSN: 0021-4868</identifier><identifier>EISSN: 1348-673X</identifier><identifier>DOI: 10.1536/ihj.34.221</identifier><identifier>PMID: 8315819</identifier><identifier>CODEN: JHEJAR</identifier><language>eng</language><publisher>Tokyo: International Heart Journal Association</publisher><subject>Adult ; Amiodarone ; Amiodarone - pharmacology ; Amiodarone - therapeutic use ; Biological and medical sciences ; Cardiomyopathy, Dilated - complications ; Defibrillation threshold ; Defibrillators, Implantable ; Drug toxicity and drugs side effects treatment ; Electrocardiography ; Humans ; Male ; Medical sciences ; Pharmacology. Drug treatments ; Tachycardia, Ventricular - complications ; Tachycardia, Ventricular - drug therapy ; Tachycardia, Ventricular - therapy ; Time Factors ; Toxicity: cardiovascular system ; Ventricular Fibrillation - therapy</subject><ispartof>Japanese Heart Journal, 1993, Vol.34(2), pp.221-226</ispartof><rights>by International Heart Journal Association</rights><rights>1993 INIST-CNRS</rights><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c473t-be5f8c35c40a909dd0709cd18903eef005ae26f04c38175ecb49abc15ad368d63</citedby></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,780,784,1883,4024,27923,27924,27925</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=4767050$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/8315819$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>GÜLDAL, Muharrem</creatorcontrib><creatorcontrib>KARAOGUZ, Remzi</creatorcontrib><creatorcontrib>AKALIN, Hakki</creatorcontrib><creatorcontrib>BAYAR, Mustafa</creatorcontrib><creatorcontrib>AKYOL, Turhan</creatorcontrib><title>Is There an Effect of Amiodarone on the Defibrillation Threshold?</title><title>Japanese Heart Journal</title><addtitle>Jpn Heart J</addtitle><description>The interaction between amiodarone and the defibrillation threshold (DFT) is still controversial. We present a case with dilated cardiomyopathy and recurrent sustained monomorphic ventricular tachycardia who received an automatic implantable cardioverter defibrillator (AICD) while under long-term amiodarone treatment. AICD implantation was performed without thoracotomy. The transvenous lead was inserted via a left subclavian vein puncture and the patch was placed on the lateral chest wall, submuscularly. At the time of implantation a 35J shock was not successful in converting ventricular fibrillation to sinus rhythm, but a 40J rescue shock was successful. After discontinuation of amiodarone, DFT measurements were repeated. Sixteen days later DFT was still higher than 34J, but 71 days later it decreased to 20J.</description><subject>Adult</subject><subject>Amiodarone</subject><subject>Amiodarone - pharmacology</subject><subject>Amiodarone - therapeutic use</subject><subject>Biological and medical sciences</subject><subject>Cardiomyopathy, Dilated - complications</subject><subject>Defibrillation threshold</subject><subject>Defibrillators, Implantable</subject><subject>Drug toxicity and drugs side effects treatment</subject><subject>Electrocardiography</subject><subject>Humans</subject><subject>Male</subject><subject>Medical sciences</subject><subject>Pharmacology. Drug treatments</subject><subject>Tachycardia, Ventricular - complications</subject><subject>Tachycardia, Ventricular - drug therapy</subject><subject>Tachycardia, Ventricular - therapy</subject><subject>Time Factors</subject><subject>Toxicity: cardiovascular system</subject><subject>Ventricular Fibrillation - therapy</subject><issn>0021-4868</issn><issn>1348-673X</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1993</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNo9kM1LxDAQxYMoun5cvAs9iAeh66T5aHqSZf1aELys4C2k6cRm6baadA_-90Z32csMzPvx3vAIuaQwpYLJO9-upoxPi4IekAllXOWyZB-HZAJQ0JwrqU7IaYwrACoLxY7JsWJUKFpNyGwRs2WLATPTZ4_OoR2zwWWztR8aE4Yes6HPxhazB3S-Dr7rzOjTadkGjO3QNffn5MiZLuLFbp-R96fH5fwlf317Xsxnr7nlJRvzGoVTlgnLwVRQNQ2UUNmGqgoYogMQBgvpgFumaCnQ1rwytaXCNEyqRrIzcrP1_QrD9wbjqNc-WkwP9Thsoi6FAsZLlcDbLWjDEGNAp7-CX5vwoynov7506kszrlNfCb7auW7qNTZ7dFdQ0q93uonWdC6Y3vq4x3gpSxCQsPkWW8XRfOJeN2H0tsO_RFpJ-E_djhS-V21rgsae_QIqZ4lh</recordid><startdate>1993</startdate><enddate>1993</enddate><creator>GÜLDAL, Muharrem</creator><creator>KARAOGUZ, Remzi</creator><creator>AKALIN, Hakki</creator><creator>BAYAR, Mustafa</creator><creator>AKYOL, Turhan</creator><general>International Heart Journal Association</general><general>Japanese Heart Journal Association</general><scope>IQODW</scope><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>1993</creationdate><title>Is There an Effect of Amiodarone on the Defibrillation Threshold?</title><author>GÜLDAL, Muharrem ; KARAOGUZ, Remzi ; AKALIN, Hakki ; BAYAR, Mustafa ; AKYOL, Turhan</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c473t-be5f8c35c40a909dd0709cd18903eef005ae26f04c38175ecb49abc15ad368d63</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1993</creationdate><topic>Adult</topic><topic>Amiodarone</topic><topic>Amiodarone - pharmacology</topic><topic>Amiodarone - therapeutic use</topic><topic>Biological and medical sciences</topic><topic>Cardiomyopathy, Dilated - complications</topic><topic>Defibrillation threshold</topic><topic>Defibrillators, Implantable</topic><topic>Drug toxicity and drugs side effects treatment</topic><topic>Electrocardiography</topic><topic>Humans</topic><topic>Male</topic><topic>Medical sciences</topic><topic>Pharmacology. Drug treatments</topic><topic>Tachycardia, Ventricular - complications</topic><topic>Tachycardia, Ventricular - drug therapy</topic><topic>Tachycardia, Ventricular - therapy</topic><topic>Time Factors</topic><topic>Toxicity: cardiovascular system</topic><topic>Ventricular Fibrillation - therapy</topic><toplevel>online_resources</toplevel><creatorcontrib>GÜLDAL, Muharrem</creatorcontrib><creatorcontrib>KARAOGUZ, Remzi</creatorcontrib><creatorcontrib>AKALIN, Hakki</creatorcontrib><creatorcontrib>BAYAR, Mustafa</creatorcontrib><creatorcontrib>AKYOL, Turhan</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Japanese Heart Journal</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>GÜLDAL, Muharrem</au><au>KARAOGUZ, Remzi</au><au>AKALIN, Hakki</au><au>BAYAR, Mustafa</au><au>AKYOL, Turhan</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Is There an Effect of Amiodarone on the Defibrillation Threshold?</atitle><jtitle>Japanese Heart Journal</jtitle><addtitle>Jpn Heart J</addtitle><date>1993</date><risdate>1993</risdate><volume>34</volume><issue>2</issue><spage>221</spage><epage>226</epage><pages>221-226</pages><issn>0021-4868</issn><eissn>1348-673X</eissn><coden>JHEJAR</coden><abstract>The interaction between amiodarone and the defibrillation threshold (DFT) is still controversial. We present a case with dilated cardiomyopathy and recurrent sustained monomorphic ventricular tachycardia who received an automatic implantable cardioverter defibrillator (AICD) while under long-term amiodarone treatment. AICD implantation was performed without thoracotomy. The transvenous lead was inserted via a left subclavian vein puncture and the patch was placed on the lateral chest wall, submuscularly. At the time of implantation a 35J shock was not successful in converting ventricular fibrillation to sinus rhythm, but a 40J rescue shock was successful. After discontinuation of amiodarone, DFT measurements were repeated. Sixteen days later DFT was still higher than 34J, but 71 days later it decreased to 20J.</abstract><cop>Tokyo</cop><pub>International Heart Journal Association</pub><pmid>8315819</pmid><doi>10.1536/ihj.34.221</doi><tpages>6</tpages><oa>free_for_read</oa></addata></record> |
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| ispartof | Japanese Heart Journal, 1993, Vol.34(2), pp.221-226 |
| issn | 0021-4868 1348-673X |
| language | eng |
| recordid | cdi_proquest_miscellaneous_75803478 |
| source | MEDLINE; J-STAGE (Japan Science & Technology Information Aggregator, Electronic) Freely Available Titles - Japanese; EZB-FREE-00999 freely available EZB journals |
| subjects | Adult Amiodarone Amiodarone - pharmacology Amiodarone - therapeutic use Biological and medical sciences Cardiomyopathy, Dilated - complications Defibrillation threshold Defibrillators, Implantable Drug toxicity and drugs side effects treatment Electrocardiography Humans Male Medical sciences Pharmacology. Drug treatments Tachycardia, Ventricular - complications Tachycardia, Ventricular - drug therapy Tachycardia, Ventricular - therapy Time Factors Toxicity: cardiovascular system Ventricular Fibrillation - therapy |
| title | Is There an Effect of Amiodarone on the Defibrillation Threshold? |
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