The neurochemical sequelae of olfactory bulbectomy

Olfactory bulbectomy in rodents leads to a variety of neurochemical sequelae involving catecholamine, indolamine, acetylcholine, amino acid and several putative neurotransmitter systems. This review summarizes the research over the last 10 years on the neurochemical consequences of olfactory bulbect...

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Veröffentlicht in:	Life Sciences 1980-05, Vol.26 (19), p.1551-1559
1. Verfasser:	Hirsch, James D.
Format:	Artikel
Sprache:	eng
Schlagworte:	Acetylcholine - metabolism Animals Aspartic Acid - metabolism Brain - enzymology Calcium - metabolism Dopamine - metabolism Epinephrine - metabolism Female gamma-Aminobutyric Acid - metabolism Glutamates - metabolism Male Mice Muscimol - pharmacology Norepinephrine - metabolism Olfactory Bulb - physiology Olfactory Bulb - surgery Ornithine Decarboxylase - metabolism Rats Receptors, Muscarinic - metabolism Serotonin - metabolism Spermine - metabolism Tyrosine 3-Monooxygenase - metabolism
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Beschreibung
Zusammenfassung:	Olfactory bulbectomy in rodents leads to a variety of neurochemical sequelae involving catecholamine, indolamine, acetylcholine, amino acid and several putative neurotransmitter systems. This review summarizes the research over the last 10 years on the neurochemical consequences of olfactory bulbectomy involving these systems. It also proposes additional experiments designed to expand our knowledge of olfactory bulb-brain neurochemical relationships.
ISSN:	0024-3205 1879-0631
DOI:	10.1016/0024-3205(80)90357-4