Immediate endocrine and metabolic consequences of traumatic quadriplegia in a young woman

Onset of paralysis by cervical spinal cord injury led immediately to temporary adrenocortical activation and, within 2 days, to sustained skin and bone breakdown. Urine cAMP was increased, blood parathyroid hormone, renin activity, and electrolytes were normal, and fluid and electrolytes balance bec...

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Veröffentlicht in:	Paraplegia 1977-11, Vol.15 (3), p.202-208
Hauptverfasser:	Claus-Walker, J, Carter, R E, Di Ferrante, M, Singh, J
Format:	Artikel
Sprache:	eng
Schlagworte:	17-Hydroxycorticosteroids - blood Adolescent Bone and Bones - metabolism Calcium - urine Collagen - metabolism Cyclic AMP - urine Female Humans Hydrocortisone - blood Hydroxyproline - metabolism Lysine - analogs & derivatives Parathyroid Hormone - blood Phosphorus - urine Quadriplegia - blood Quadriplegia - metabolism Quadriplegia - urine Renin - blood Syndrome Water-Electrolyte Imbalance - etiology
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Beschreibung
Zusammenfassung:	Onset of paralysis by cervical spinal cord injury led immediately to temporary adrenocortical activation and, within 2 days, to sustained skin and bone breakdown. Urine cAMP was increased, blood parathyroid hormone, renin activity, and electrolytes were normal, and fluid and electrolytes balance became negative during the initial 6 days of paralysis.
ISSN:	0031-1758 1362-4393 1476-5624
DOI:	10.1038/sc.1977.30