γ-Glutamyltranspeptidase–Deficient Knockout Mice as a Model to Study the Relationship Between Glutathione Status, Mitochondrial Function, and Cellular Function

γ-Glutamyltranspeptidase (GGT)-deficient mice (GGT−/−) display chronic glutathione (GSH) deficiency, growth retardation, and die at a young age (

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Bibliographische Detailangaben
Veröffentlicht in:	Hepatology (Baltimore, Md.) Md.), 2000-10, Vol.32 (4), p.740-749
Hauptverfasser:	Will, Yvonne, Fischer, Kay A, Horton, Robert A, Kaetzel, Rhonda S, Brown, Marda K, Hedstrom, Olaf, Lieberman, Michael W, Reed, Donald J
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenosine Diphosphate - analysis Adenosine Triphosphate - analysis Adenosine Triphosphate - biosynthesis Animals Biological and medical sciences gamma-Glutamyltransferase - deficiency Gastroenterology. Liver. Pancreas. Abdomen Glutathione - analysis Glutathione - metabolism Liver - cytology Liver - ultrastructure Liver. Biliary tract. Portal circulation. Exocrine pancreas Medical sciences Mice Mice, Knockout Microscopy, Electron Mitochondria, Liver - physiology Other diseases. Semiology Oxygen Consumption
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Beschreibung
Zusammenfassung:	γ-Glutamyltranspeptidase (GGT)-deficient mice (GGT−/−) display chronic glutathione (GSH) deficiency, growth retardation, and die at a young age (
ISSN:	0270-9139 1527-3350
DOI:	10.1053/jhep.2000.17913