ING1b decreases cell proliferation through p53-dependent and -independent mechanisms

ING1b can stimulate cell cycle arrest, repair, senescence, and apoptosis. The actions of ING1b are attributed to its activation of the tumor suppressor p53. Here we investigate the more subtle effects of ING1b on the cell cycle and DNA damage responses in the absence of p53. To this end, we have gen...

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Veröffentlicht in:FEBS letters 2003-10, Vol.553 (3), p.277-285
Hauptverfasser: Tsang, Fan Cheung, Po, Lai See, Leung, Ka Man, Lau, Anita, Siu, Wai Yi, Poon, Randy Y.C.
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Sprache:eng
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