The proapoptotic BCL-2 family member BIM mediates motoneuron loss in a model of amyotrophic lateral sclerosis

Gespeichert in:

Bibliographische Detailangaben
Veröffentlicht in:	Cell death and differentiation 2007-07, Vol.14 (7), p.1386-1389
Hauptverfasser:	Hetz, C, Thielen, P, Fisher, J, Pasinelli, P, Brown, R H, Korsmeyer, S, Glimcher, L
Format:	Artikel
Sprache:	eng
Schlagworte:	Amyotrophic Lateral Sclerosis - metabolism Amyotrophic Lateral Sclerosis - pathology Amyotrophic Lateral Sclerosis - physiopathology Animals Apoptosis Apoptosis - genetics Apoptosis Regulatory Proteins - genetics Apoptosis Regulatory Proteins - metabolism Bcl-2-Like Protein 11 bcl-X Protein - genetics bcl-X Protein - metabolism Biochemistry Biomedical and Life Sciences Cell Biology Cell Cycle Analysis Disease Models, Animal Down-Regulation - genetics Humans In Situ Nick-End Labeling letter-to-the-editor Life Sciences Membrane Proteins - genetics Membrane Proteins - metabolism Mice Mice, Knockout Mice, Transgenic Motor Neurons - metabolism Motor Neurons - pathology Mutation - genetics Nerve Degeneration - metabolism Nerve Degeneration - pathology Nerve Degeneration - physiopathology Proto-Oncogene Proteins - genetics Proto-Oncogene Proteins - metabolism Proto-Oncogene Proteins c-bcl-2 - genetics Proto-Oncogene Proteins c-bcl-2 - metabolism Stem Cells Superoxide Dismutase - genetics Superoxide Dismutase - metabolism Superoxide Dismutase-1 Up-Regulation - genetics
Online-Zugang:	Volltext
Tags:	Tag hinzufügen Keine Tags, Fügen Sie den ersten Tag hinzu!