The type III secretion effector XopXccN of Xanthomonas campestris pv. campestris is required for full virulence

XopN was originally identified from Xanthomonas campestris pv. vesicatoria as an effector translocated into plant cells via the type III secretion system (T3SS), and is required for pathogenicity. We report here that the xopN homologue in the X. campestris pv. campestris genome, named xopXccN, also...

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Veröffentlicht in:	Research in microbiology 2008-04, Vol.159 (3), p.216-220
Hauptverfasser:	Jiang, Bo-Le, He, Yong-Qiang, Cen, Wei-Jian, Wei, Hong-Yu, Jiang, Guo-Feng, Jiang, Wei, Hang, Xiao-Hong, Feng, Jia-Xun, Lu, Guang-Tao, Tang, Dong-Jie, Tang, Ji-Liang
Format:	Artikel
Sprache:	eng
Schlagworte:	Bacterial Proteins - genetics Bacterial Proteins - metabolism Bacteriology Biological and medical sciences Biological Transport Fundamental and applied biological sciences. Psychology Genome, Bacterial Microbiology Miscellaneous Plant Diseases - microbiology Transcription Factors - metabolism Type III secretion effector Virulence Xanthomonas campestris Xanthomonas campestris - genetics Xanthomonas campestris - metabolism Xanthomonas campestris - pathogenicity Xanthomonas campestris pv. campestris XopXccN
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Zusammenfassung:	XopN was originally identified from Xanthomonas campestris pv. vesicatoria as an effector translocated into plant cells via the type III secretion system (T3SS), and is required for pathogenicity. We report here that the xopN homologue in the X. campestris pv. campestris genome, named xopXccN, also encodes a T3SS effector and is required for full virulence. We further demonstrate that expression of xopXccN is positively regulated by the key hrp (hypersensitive response and pathogenicity) regulators HrpG and HrpX.
ISSN:	0923-2508 1769-7123
DOI:	10.1016/j.resmic.2007.12.004