Ca2+-Calmodulin–Dependent Protein Kinase Phosphorylation of Ryanodine Receptor May Contribute to the β-Adrenergic Regulation of Myocardial Contractility Independently of Increases in Heart Rate

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Veröffentlicht in:	Circulation research 2008-07, Vol.103 (1), p.e10-e11
Hauptverfasser:	Mattiazzi, Alicia, Vittone, Leticia, Mundiña-Weilenmann, Cecilia
Format:	Artikel
Sprache:	eng
Schlagworte:	Amino Acid Substitution Animals Cyclic AMP-Dependent Protein Kinases - genetics Cyclic AMP-Dependent Protein Kinases - metabolism Electrophysiologic Techniques, Cardiac Isoproterenol - pharmacology Mice Myocardial Contraction - physiology Myocardium - enzymology Phosphorylation Ryanodine Receptor Calcium Release Channel - genetics Ryanodine Receptor Calcium Release Channel - metabolism Serine - metabolism Sympathetic Nervous System - metabolism
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