Lymphadenopathy Due to Amyloidosis Secondary to Sjögren Syndrome and Systemic Lupus Erythematosus Detected by F-18 FDG PET
We report a case showing lymphadenopathy due to amyloidosis secondary to Sjögren syndrome and systemic lupus erythematosus (SLE). The amyloid lymphadenopathy was detected by positron emission tomography (PET) as faint but significant uptake of F-18 fluorodeoxyglucose (FDG). Lymph node amyloidoses af...
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Veröffentlicht in: | Clinical nuclear medicine 2007-11, Vol.32 (11), p.881-882 |
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Hauptverfasser: | , , , , , , , , , |
Format: | Artikel |
Sprache: | eng |
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Zusammenfassung: | We report a case showing lymphadenopathy due to amyloidosis secondary to Sjögren syndrome and systemic lupus erythematosus (SLE). The amyloid lymphadenopathy was detected by positron emission tomography (PET) as faint but significant uptake of F-18 fluorodeoxyglucose (FDG). Lymph node amyloidoses after SLE are probably unusual, and those secondary to Sjögren syndrome remain rare. FDG-PET findings of amyloidosis have been exclusively described with pulmonary amyloidomas. When a patient with a connective tissue disease develops significant FDG uptake in the lymph nodes, the possibility of amyloidosis should be considered. |
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ISSN: | 0363-9762 1536-0229 |
DOI: | 10.1097/RLU.0b013e318156bb56 |