Nephrotic syndrome and IgA nephropathy in polycystic kidney disease

A 70-year-old man with polycystic kidney disease developed nephrotic syndrome, deteriorating to renal insufficiency. Histological examination revealed IgA nephropathy. With treatment of prednisolone, an angiotensin-converting enzyme inhibitor, and an angiotensin II receptor-blocker, his proteinuria...

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Veröffentlicht in:	Clinical and experimental nephrology 2006-06, Vol.10 (2), p.136-139
Hauptverfasser:	Hiura, Toru, Yamazaki, Hajime, Saeki, Takako, Kawabe, Shotetsu, Ueno, Mitsuhiro, Nishi, Shinichi, Miyamura, Shoji, Gejyo, Fumitake
Format:	Artikel
Sprache:	eng
Schlagworte:	Aged Angiotensin Angiotensin II Angiotensin-converting enzyme inhibitors Biopsy Dipyridamole - therapeutic use Glomerulonephritis, IGA - drug therapy Glomerulonephritis, IGA - etiology Humans IgA nephropathy Immunoglobulin A Kidney - pathology Kidney diseases Losartan - therapeutic use Male Nephrotic syndrome Nephrotic Syndrome - drug therapy Nephrotic Syndrome - etiology Peptidyl-dipeptidase A Polycystic kidney Polycystic Kidney, Autosomal Dominant - complications Polycystic Kidney, Autosomal Dominant - drug therapy Polycystic Kidney, Autosomal Dominant - pathology Prednisolone Prednisolone - therapeutic use Proteinuria Renal failure Renal function Renal insufficiency Thiazepines - therapeutic use
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Zusammenfassung:	A 70-year-old man with polycystic kidney disease developed nephrotic syndrome, deteriorating to renal insufficiency. Histological examination revealed IgA nephropathy. With treatment of prednisolone, an angiotensin-converting enzyme inhibitor, and an angiotensin II receptor-blocker, his proteinuria markedly decreased and renal function was stabilized. This case supports the idea that renal biopsy is needed in patients with polycystic kidney disease with nephrotic-range proteinuria, for appropriate treatment and prevention of renal failure.
ISSN:	1342-1751 1437-7799
DOI:	10.1007/s10157-005-0403-6