Rapid Action of Oestrogen in Luteinising Hormone-Releasing Hormone Neurones: The Role of GPR30

Previously, we have shown that 17β‐oestradiol (E2) induces an increase in firing activity and modifies the pattern of intracellular calcium ([Ca2+]i) oscillations with a latency

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Bibliographische Detailangaben
Veröffentlicht in:	Journal of neuroendocrinology 2009-04, Vol.21 (4), p.316-321
Hauptverfasser:	Terasawa, E., Noel, S. D., Keen, K. L.
Format:	Artikel
Sprache:	eng
Schlagworte:	Action Potentials - drug effects Animals Biological and medical sciences Calcium - metabolism Cell Membrane - physiology Estradiol - pharmacology Estrogen Receptor beta - metabolism Estrogens - metabolism Estrogens - pharmacology Fundamental and applied biological sciences. Psychology GnRH neurones GPCR GPR30 Luteinizing Hormone - metabolism Neurons - drug effects Neurons - metabolism Periodicity Primates rapid action of oestrogen Receptors, G-Protein-Coupled - metabolism Time Factors Vertebrates: endocrinology
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Beschreibung
Zusammenfassung:	Previously, we have shown that 17β‐oestradiol (E2) induces an increase in firing activity and modifies the pattern of intracellular calcium ([Ca2+]i) oscillations with a latency
ISSN:	0953-8194 1365-2826
DOI:	10.1111/j.1365-2826.2009.01839.x