The Conundrum Posed by Cellular Heterogeneity in Analysis of Human Neuroblastoma

200, Department of Biological Sciences, Fordham University, 441 E. FordhamRd., Bronx, NY 10458 (e-mail: rross@fordham.edu).DOI: 10.1093/jnci/djh262Journal of the National Cancer Institute, Vol. 96, No. 16, Oxford UniversityPress 2004, all rights reserved.1192 EDITORIALS Journal of the National Cance...

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Veröffentlicht in:	JNCI : Journal of the National Cancer Institute 2004-08, Vol.96 (16), p.1192-1193
Hauptverfasser:	Ross, Robert A., Spengler, Barbara A.
Format:	Artikel
Sprache:	eng
Schlagworte:	Cell Line, Tumor Clone Cells Cluster Analysis DNA Methylation DNA, Neoplasm - metabolism Gene Amplification Gene Expression Regulation, Neoplastic Gene Silencing Humans Multigene Family N-Myc Proto-Oncogene Protein Neuroblastoma - genetics Nuclear Proteins - genetics Oncogene Proteins - genetics Promoter Regions, Genetic
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Zusammenfassung:	200, Department of Biological Sciences, Fordham University, 441 E. FordhamRd., Bronx, NY 10458 (e-mail: rross@fordham.edu).DOI: 10.1093/jnci/djh262Journal of the National Cancer Institute, Vol. 96, No. 16, Oxford UniversityPress 2004, all rights reserved.1192 EDITORIALS Journal of the National Cancer Institute, Vol. 96, No. 16, August 18, 2004(caspase-8, FLIP, DcR1, DcR2, DR4, and DR5) were hypermethylated in the majority of cell lines. In tumors, only the loss of
ISSN:	0027-8874 1460-2105
DOI:	10.1093/jnci/djh262