Cell cycle control in cancer
Cancer is a group of diseases in which cells divide continuously and excessively. Cell division is tightly regulated by multiple evolutionarily conserved cell cycle control mechanisms, to ensure the production of two genetically identical cells. Cell cycle checkpoints operate as DNA surveillance mec...
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Veröffentlicht in: | Nature reviews. Molecular cell biology 2022-01, Vol.23 (1), p.74-88 |
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description | Cancer is a group of diseases in which cells divide continuously and excessively. Cell division is tightly regulated by multiple evolutionarily conserved cell cycle control mechanisms, to ensure the production of two genetically identical cells. Cell cycle checkpoints operate as DNA surveillance mechanisms that prevent the accumulation and propagation of genetic errors during cell division. Checkpoints can delay cell cycle progression or, in response to irreparable DNA damage, induce cell cycle exit or cell death. Cancer-associated mutations that perturb cell cycle control allow continuous cell division chiefly by compromising the ability of cells to exit the cell cycle. Continuous rounds of division, however, create increased reliance on other cell cycle control mechanisms to prevent catastrophic levels of damage and maintain cell viability. New detailed insights into cell cycle control mechanisms and their role in cancer reveal how these dependencies can be best exploited in cancer treatment.
This Review discusses our current understanding of cell cycle regulation, the functions of cell cycle checkpoints and how disruption of these finely tuned mechanisms is associated with cancer. Insights into these regulatory mechanisms are creating new opportunities for the treatment of cancer. |
doi_str_mv | 10.1038/s41580-021-00404-3 |
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Molecular cell biology</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Matthews, Helen K.</au><au>Bertoli, Cosetta</au><au>de Bruin, Robertus A. M.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Cell cycle control in cancer</atitle><jtitle>Nature reviews. Molecular cell biology</jtitle><stitle>Nat Rev Mol Cell Biol</stitle><addtitle>Nat Rev Mol Cell Biol</addtitle><date>2022-01-01</date><risdate>2022</risdate><volume>23</volume><issue>1</issue><spage>74</spage><epage>88</epage><pages>74-88</pages><issn>1471-0072</issn><eissn>1471-0080</eissn><abstract>Cancer is a group of diseases in which cells divide continuously and excessively. Cell division is tightly regulated by multiple evolutionarily conserved cell cycle control mechanisms, to ensure the production of two genetically identical cells. Cell cycle checkpoints operate as DNA surveillance mechanisms that prevent the accumulation and propagation of genetic errors during cell division. Checkpoints can delay cell cycle progression or, in response to irreparable DNA damage, induce cell cycle exit or cell death. Cancer-associated mutations that perturb cell cycle control allow continuous cell division chiefly by compromising the ability of cells to exit the cell cycle. Continuous rounds of division, however, create increased reliance on other cell cycle control mechanisms to prevent catastrophic levels of damage and maintain cell viability. New detailed insights into cell cycle control mechanisms and their role in cancer reveal how these dependencies can be best exploited in cancer treatment.
This Review discusses our current understanding of cell cycle regulation, the functions of cell cycle checkpoints and how disruption of these finely tuned mechanisms is associated with cancer. Insights into these regulatory mechanisms are creating new opportunities for the treatment of cancer.</abstract><cop>London</cop><pub>Nature Publishing Group UK</pub><pmid>34508254</pmid><doi>10.1038/s41580-021-00404-3</doi><tpages>15</tpages><orcidid>https://orcid.org/0000-0001-9957-1409</orcidid><orcidid>https://orcid.org/0000-0001-5684-4630</orcidid><oa>free_for_read</oa></addata></record> |
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subjects | 631/337/1427/2567 631/337/641/2187 631/67 631/67/1244 631/80/641 Animals Biochemistry Biomedical and Life Sciences Cancer Cancer Research Cell Biology Cell cycle Cell Cycle Checkpoints Cell death Cell division Cell viability Damage prevention Deoxyribonucleic acid Developmental Biology DNA DNA damage DNA Damage - genetics DNA Replication - genetics Humans Life Sciences Mutation Neoplasms - genetics Neoplasms - pathology Neoplasms - therapy Oncogenes Regulatory mechanisms (biology) Review Article Spindle Apparatus - metabolism Stem Cells |
title | Cell cycle control in cancer |
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