Antioxidants & bronchopulmonary dysplasia: Beating the system or beating a dead horse?

Preterm birth is a primary cause of worldwide childhood mortality. Bronchopulmonary dysplasia, characterized by impaired alveolar and lung vascular development, affects 25–50% of extremely low birth weight (BW;

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Bibliographische Detailangaben
Veröffentlicht in:	Free radical biology & medicine 2019-10, Vol.142, p.138-145
Hauptverfasser:	Ofman, Gaston, Tipple, Trent E.
Format:	Artikel
Sprache:	eng
Schlagworte:	Antioxidants - therapeutic use Bronchopulmonary Dysplasia - genetics Bronchopulmonary Dysplasia - metabolism Bronchopulmonary Dysplasia - physiopathology Bronchopulmonary Dysplasia - therapy Child Dietary Supplements Glutathione - administration & dosage Glutathione - metabolism Humans Infant, Newborn Infant, Premature Infant, Very Low Birth Weight Intensive Care Units, Neonatal Lung - drug effects Lung - metabolism Lung - physiopathology NF-E2-Related Factor 2 - agonists NF-E2-Related Factor 2 - genetics NF-E2-Related Factor 2 - metabolism Oxidative Stress - drug effects Precision Medicine - methods Reactive Oxygen Species - antagonists & inhibitors Reactive Oxygen Species - metabolism Selenium - administration & dosage Selenium - metabolism Thioredoxins - agonists Thioredoxins - genetics Thioredoxins - metabolism
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Beschreibung
Zusammenfassung:	Preterm birth is a primary cause of worldwide childhood mortality. Bronchopulmonary dysplasia, characterized by impaired alveolar and lung vascular development, affects 25–50% of extremely low birth weight (BW;
ISSN:	0891-5849 1873-4596
DOI:	10.1016/j.freeradbiomed.2019.01.038