Intraluminal pressure of uterine balloon tamponade in the management of severe post‐partum hemorrhage

Aim Intrauterine balloon tamponade has been increasingly used for the management of post‐partum hemorrhage (PPH) in recent years. However, data on the precise mechanisms and pressure required for the balloon tamponade are scanty in the literature. This study aims to review the intraluminal pressure...

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Veröffentlicht in:The journal of obstetrics and gynaecology research 2018-05, Vol.44 (5), p.914-921
Hauptverfasser: Kong, Choi Wah, To, William W. K.
Format: Artikel
Sprache:eng
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Zusammenfassung:Aim Intrauterine balloon tamponade has been increasingly used for the management of post‐partum hemorrhage (PPH) in recent years. However, data on the precise mechanisms and pressure required for the balloon tamponade are scanty in the literature. This study aims to review the intraluminal pressure (ILP) generated by the Bakri intrauterine balloon that is necessary to produce a ‘positive tamponade test’ during severe PPH. Methods This was a prospective cohort study. The ILP of the Bakri balloon was measured using a manometer after a positive tamponade test was clinically achieved during severe PPH (blood loss >1 L). The patient's blood pressure was recorded, and ultrasound scan was performed to verify the position of the balloon and the presence of forward flow in the uterine arteries. The main outcome measure is the ILP of the Bakri balloon required to achieve a positive tamponade test. Results Twenty patients were included for final analysis. The net ILP measured ranged from 67 to 92 mmHg, and this pressure was lower than the concurrent systolic pressure in all cases. Color Doppler confirmed positive forward flow in the uterine vessels in all cases. There were no differences in the pressure measured with the balloon position, and there was no relationship between the volumes of saline infused and the net pressure. Conclusion A positive tamponade test in an intrauterine balloon is probably achieved by local compression pressure exerted on the vasculature of the placental bed rather than by generating an ILP exceeding systemic blood pressure or by occlusion of flow to the uterine arteries.
ISSN:1341-8076
1447-0756
DOI:10.1111/jog.13596