Occupational manganese neurotoxicity provoked by hepatitis C

Manganese neurotoxicity developed in a highly exposed worker after asymptomatic, moderate hepatic dysfunction from hepatitis C infection. Antiviral therapy was accompanied by resolution of increased blood manganese levels and neurologic improvement. Even asymptomatic hepatic dysfunction may impair m...

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Veröffentlicht in:	Neurology 2006-07, Vol.67 (2), p.322-323
Hauptverfasser:	SCHAUMBURG, Herbert H, HERSKOVITZ, Steven, CASSANO, Victoria A
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Biological and medical sciences Female Hepatitis C - complications Hepatitis C - diagnosis Human viral diseases Humans Infectious diseases Manganese Poisoning - diagnosis Manganese Poisoning - etiology Medical sciences Metallurgy Neurology Neuropharmacology Neuroprotective agent Occupational Diseases - diagnosis Occupational Diseases - etiology Occupational Exposure - adverse effects Pharmacology. Drug treatments Viral diseases Viral hepatitis
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Beschreibung
Zusammenfassung:	Manganese neurotoxicity developed in a highly exposed worker after asymptomatic, moderate hepatic dysfunction from hepatitis C infection. Antiviral therapy was accompanied by resolution of increased blood manganese levels and neurologic improvement. Even asymptomatic hepatic dysfunction may impair manganese clearance and place highly exposed persons at risk for toxicity.
ISSN:	0028-3878 1526-632X
DOI:	10.1212/01.wnl.0000225179.21282.58