Light-emitting diode therapy reduces persistent inflammatory pain: Role of interleukin 10 and antioxidant enzymes

Highlights • LEDT inhibited mechanical and thermal hiperalgesia. • LEDT increased the levels of IL-10. • LEDT induced an increase in both SOD and CAT activity.

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Bibliographische Detailangaben
Veröffentlicht in:	Neuroscience 2016-06, Vol.324, p.485-495
Hauptverfasser:	Martins, D.F, Turnes, B.L, Cidral-Filho, F.J, Bobinski, F, Rosas, R.F, Danielski, L.G, Petronilho, F, Santos, A.R.S
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Antioxidants - metabolism Catalase - metabolism Disease Models, Animal Freund's Adjuvant Hot Temperature Hyperalgesia - metabolism Hyperalgesia - therapy inflammation Inflammation - metabolism Inflammation - therapy Interleukin-10 - metabolism Interleukin-1beta - metabolism low-level light therapy Male Mice Neurology Oxidative Stress - physiology Pain - metabolism Pain Management - instrumentation Pain Management - methods persistent pain Phototherapy - instrumentation Phototherapy - methods Superoxide Dismutase - metabolism Thiobarbituric Acid Reactive Substances - metabolism Touch Treatment Outcome Tumor Necrosis Factor-alpha - metabolism
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Beschreibung
Zusammenfassung:	Highlights • LEDT inhibited mechanical and thermal hiperalgesia. • LEDT increased the levels of IL-10. • LEDT induced an increase in both SOD and CAT activity.
ISSN:	0306-4522 1873-7544
DOI:	10.1016/j.neuroscience.2016.03.035