Vitamin E and β-carotene protect against ethanol combined with ischemia in an embryonic rat hippocampal culture model of fetal alcohol syndrome

Neurodevelopmental damage can occur as a result of in utero exposure to alcohol. Oxidative stress processes are one of many proposed mechanisms thought to contribute to nervous system dysfunction characterized in fetal alcohol syndrome (FAS). Therefore, this study examined neuroprotective effects of...

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Veröffentlicht in:	Neuroscience letters 1999-03, Vol.263 (2), p.189-192
Hauptverfasser:	Mitchell, J.Jean, Paiva, Michael, Heaton, Marieta B
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Antioxidants Antioxidants - pharmacology beta Carotene - pharmacology Biological and medical sciences Brain Ischemia Cell Survival - drug effects Cells, Cultured Embryo, Mammalian Ethanol Ethanol - toxicity Fetal Alcohol Spectrum Disorders - physiopathology Fetal Alcohol Spectrum Disorders - prevention & control Hippocampus Hippocampus - cytology Hypoglycemia Ischemia Medical sciences Neurons - cytology Neurons - drug effects Neurons - physiology Neuropharmacology Neuroprotective agent Neuroprotective Agents - pharmacology Oxidative Stress - drug effects Oxidative Stress - physiology Pharmacology. Drug treatments Rats Rats, Long-Evans Vitamin E - pharmacology Vitamin E, β-carotene
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