Modulation of local field potential power of the subthalamic nucleus during isometric force generation in patients with Parkinson’s disease

Highlights ► STN local field potential (LFP) power is modulated during an isometric force task. ► This modulation of STN LFP power occurs in the upper beta- and gamma-band. ► It is not associated with a task-related modulation of Granger-causality.

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Bibliographische Detailangaben
Veröffentlicht in:	Neuroscience 2013-06, Vol.240, p.106-116
Hauptverfasser:	Florin, E, Dafsari, H.S, Reck, C, Barbe, M.T, Pauls, K.A.M, Maarouf, M, Sturm, V, Fink, G.R, Timmermann, L
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Aged Analysis of Variance basal ganglia Biological and medical sciences closed-loop stimulation deep brain stimulation (DBS) Degenerative and inherited degenerative diseases of the nervous system. Leukodystrophies. Prion diseases Electromyography Evoked Potentials - physiology Female Fourier Analysis Fundamental and applied biological sciences. Psychology Granger-causality Humans Image Processing, Computer-Assisted intraoperative recordings Isometric Contraction - physiology Magnetic Resonance Imaging Male Medical sciences Middle Aged Motor control and motor pathways. Reflexes. Control centers of vegetative functions. Vestibular system and equilibration Nerve Net - blood supply Nerve Net - physiology Nervous system (semeiology, syndromes) Nervous system as a whole Neurology Oxygen - blood Parkinson Disease - pathology Parkinson Disease - physiopathology squared partial directed coherence Subthalamic Nucleus - blood supply Subthalamic Nucleus - diagnostic imaging Subthalamic Nucleus - physiopathology Tomography, X-Ray Computed Vertebrates: nervous system and sense organs
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Beschreibung
Zusammenfassung:	Highlights ► STN local field potential (LFP) power is modulated during an isometric force task. ► This modulation of STN LFP power occurs in the upper beta- and gamma-band. ► It is not associated with a task-related modulation of Granger-causality.
ISSN:	0306-4522 1873-7544
DOI:	10.1016/j.neuroscience.2013.02.043