TRPV1 Channels: Not So Inactive on the ER

In this issue of Neuron, Wong et al. (2014) report a remarkable evolutionarily conserved role for the Drosophila TRPV1 homolog Inactive controlling synaptic growth at larval neuromuscular junctions by facilitating Ca2+ release from the endoplasmic reticulum. In this issue of Neuron, Wong et al. (201...

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Veröffentlicht in:	Neuron (Cambridge, Mass.) Mass.), 2014-11, Vol.84 (4), p.659-661
Hauptverfasser:	Imler, Elliot, Zinsmaier, Konrad E.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Calcium - metabolism Drosophila Drosophila Proteins - metabolism Ion Channels - metabolism Motor Neurons - metabolism Neuromuscular Junction - metabolism Presynaptic Terminals - metabolism Synaptic Transmission - physiology TRPV Cation Channels - metabolism
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Beschreibung
Zusammenfassung:	In this issue of Neuron, Wong et al. (2014) report a remarkable evolutionarily conserved role for the Drosophila TRPV1 homolog Inactive controlling synaptic growth at larval neuromuscular junctions by facilitating Ca2+ release from the endoplasmic reticulum. In this issue of Neuron, Wong et al. (2014) report a remarkable evolutionarily conserved role for the Drosophila TRPV1 homolog Inactive controlling synaptic growth at larval neuromuscular junctions by facilitating Ca2+ release from the endoplasmic reticulum.
ISSN:	0896-6273 1097-4199
DOI:	10.1016/j.neuron.2014.10.052