Acetylsalicylic acid enhances the anti-inflammatory effect of fluoxetine through inhibition of NF-κB, p38-MAPK and ERK1/2 activation in lipopolysaccharide-induced BV-2 microglia cells

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Veröffentlicht in:Neuroscience 2014-09, Vol.275, p.296-304
Hauptverfasser: Yang, J.M, Rui, B.B, Chen, C, Chen, H, Xu, T.J, Xu, W.P, Wei, W
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container_end_page 304
container_issue
container_start_page 296
container_title Neuroscience
container_volume 275
creator Yang, J.M
Rui, B.B
Chen, C
Chen, H
Xu, T.J
Xu, W.P
Wei, W
description Graphical abstract
doi_str_mv 10.1016/j.neuroscience.2014.06.016
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subjects Animals
Anti-Inflammatory Agents - pharmacology
Antidepressive Agents, Second-Generation - pharmacology
Aspirin - pharmacology
Blotting, Western
Cell Line
Cell Survival - drug effects
Enzyme-Linked Immunosorbent Assay
Fluoxetine - pharmacology
Immunohistochemistry
Inflammation - chemically induced
Inflammation - metabolism
Lipopolysaccharides - toxicity
MAP Kinase Signaling System - drug effects
Mice
Microglia - drug effects
Microglia - metabolism
Neurology
NF-kappa B - drug effects
NF-kappa B - metabolism
p38 Mitogen-Activated Protein Kinases - drug effects
p38 Mitogen-Activated Protein Kinases - metabolism
Signal Transduction - drug effects
title Acetylsalicylic acid enhances the anti-inflammatory effect of fluoxetine through inhibition of NF-κB, p38-MAPK and ERK1/2 activation in lipopolysaccharide-induced BV-2 microglia cells
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