Altered aquaporins in the brains of mice submitted to intermittent hypoxia model of sleep apnea

Highlights ► Intermittent hypoxia affects the expression of brain aquaporins. ► Mice exposed to intermittent hypoxia presented increased brain water content. ► Aquaporins might play a role in the pathophysiology of sleep apnea syndromes.

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Bibliographische Detailangaben
Veröffentlicht in:	Respiratory physiology & neurobiology 2013-01, Vol.185 (2), p.217-221
Hauptverfasser:	Baronio, Diego, Martinez, Denis, Fiori, Cintia Zappe, Bambini-Junior, Victorio, Forgiarini, Luiz Felipe, Pase da Rosa, Darlan, Kim, Lenise Jihe, Cerski, Marcelle Reesink
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Aquaporins Aquaporins - metabolism Brain - metabolism Disease Models, Animal Edema Glial Fibrillary Acidic Protein - metabolism Hypoxia - complications Intermittent hypoxia Male Medical Education Mice Mice, Inbred C57BL Nerve Growth Factors - metabolism Organ Size OSA Pulmonary/Respiratory S100 Calcium Binding Protein beta Subunit S100 Proteins - metabolism Sleep Apnea Syndromes - etiology Sleep Apnea Syndromes - pathology Statistics, Nonparametric Water - metabolism
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Beschreibung
Zusammenfassung:	Highlights ► Intermittent hypoxia affects the expression of brain aquaporins. ► Mice exposed to intermittent hypoxia presented increased brain water content. ► Aquaporins might play a role in the pathophysiology of sleep apnea syndromes.
ISSN:	1569-9048 1878-1519
DOI:	10.1016/j.resp.2012.10.012