Renal ammonia metabolic response in the rat to repeated ethanol loading

Renal ammonia metabolic response in the rat to repeated ethanol loading

Lactate dehydrogenase (LDH), succinate dehydrogenase (SDH), aspartate aminotransferase (AAT), glutamate dehydrogenase (GDH), AMP deaminase, ornithine transcarbamylase (OTC), arginase and glutamine synthetase (GS) activities were increased in the kidney of the rat during repeated ethanol loading. The...

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Veröffentlicht in:Toxicology letters 1984-01, Vol.22 (3), p.339-342
Hauptverfasser: Mohanachari, V., Reddy, M.M., Indira, K.
Format: Artikel
Sprache:eng
Schlagworte:
Alcoholism and acute alcohol poisoning
Ammonia
Ammonia - metabolism
ammoniagenesis
Animals
Biological and medical sciences
Citric Acid Cycle
Deamination
Ethanol - toxicity
ethanol-caused hyperammonemia
glutamine
Inactivation, Metabolic
Male
Medical sciences
Rats
Rats, Inbred Strains
Toxicology
urea cycle enzymes
urea synthesis
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Zusammenfassung:Lactate dehydrogenase (LDH), succinate dehydrogenase (SDH), aspartate aminotransferase (AAT), glutamate dehydrogenase (GDH), AMP deaminase, ornithine transcarbamylase (OTC), arginase and glutamine synthetase (GS) activities were increased in the kidney of the rat during repeated ethanol loading. The significance of these findings is discussed.
ISSN:0378-4274
1879-3169
DOI:10.1016/0378-4274(84)90111-5