Acute neurogenic pulmonary edema following electroconvulsive therapy: a case report

Abstract Objective We report the case of a 47-year-old man with depression who developed acute dyspnea, hypoxemia, and mild hemoptysis after electroconvulsive therapy (ECT). Method Intravenous carbazochrome sodium sulfate hydrate as a hemostatic drug (100 mg/day) was prescribed for 2 days. On the da...

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Veröffentlicht in:General hospital psychiatry 2012-11, Vol.34 (6), p.703.e9-703.e11
Hauptverfasser: Takahashi, Tohru, M.D., Ph.D, Kinoshita, Kuni, M.D, Fuke, Tomonori, M.D, Urushihata, Kazuhisa, M.D, Kawamata, Tomoyuki, M.D., Ph.D, Yanagisawa, Shin, M.D, Kaneko, Tomoki, M.D, Washizuka, Shinsuke, M.D., Ph.D, Hanihara, Tokiji, M.D., Ph.D, Amano, Naoji, M.D., Ph.D
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Sprache:eng
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Zusammenfassung:Abstract Objective We report the case of a 47-year-old man with depression who developed acute dyspnea, hypoxemia, and mild hemoptysis after electroconvulsive therapy (ECT). Method Intravenous carbazochrome sodium sulfate hydrate as a hemostatic drug (100 mg/day) was prescribed for 2 days. On the day of ECT, oxygen inhalation (4 L/min) was continued, and SpO2 was maintained at 94–96%. Results Chest radiography showed improvement in alveolar infiltration. Chest CT 6 days after ECT also confirmed the disappearance of ground glass opacities in the lung fields. Conclusion(s) NPE is lifethreatening and should be recognized as an uncommon adverse event associated with ECT.
ISSN:0163-8343
1873-7714
DOI:10.1016/j.genhosppsych.2012.03.004