High sodium intake adversely affects oxidative-inflammatory response, cardiac remodelling and mortality after myocardial infarction

Highlights ► The effect of sodium intake on the outcome after myocardial infarction is unknown. ► High sodium intake favors inflammation and ventricular remodeling. ► High sodium intakers have higher short and long-term mortality after myocardial infarction.

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Bibliographische Detailangaben
Veröffentlicht in:	Atherosclerosis 2012-05, Vol.222 (1), p.284-291
Hauptverfasser:	Costa, Ana Paula R, de Paula, Rafaela C.S, Carvalho, Guilherme F, Araújo, Juliana P, Andrade, Joalbo M, de Almeida, Osorio L.R, de Faria, Eliana C, Freitas, Wladimir M, Coelho, Otavio R, Ramires, Jose A.F, Quinaglia e Silva, Jose C, Sposito, Andrei C
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult animal models atherosclerosis Atherosclerosis (general aspects, experimental research) B-type natriuretic peptide Biological and medical sciences Blood and lymphatic vessels C-reactive protein C-Reactive Protein - metabolism Cardiology. Vascular system Cardiovascular Coronary heart disease Creatine Kinase, MB Form - blood Dinoprost - analogs & derivatives Dinoprost - blood Female Follow-Up Studies food intake Heart Humans Inflammation interleukin-2 Interleukin-2 - blood magnetic resonance imaging Male Medical sciences Middle Aged Mortality Myocardial infarction Myocardial Infarction - blood Myocardial Infarction - mortality Myocardial Infarction - physiopathology Natriuretic Peptide, Brain - blood natriuretic peptides oxidative stress Oxidative Stress - physiology patients questionnaires Sodium Sodium - administration & dosage Sodium - adverse effects tumor necrosis factor-alpha Tumor Necrosis Factor-alpha - blood Ventricular Remodeling - physiology
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Beschreibung
Zusammenfassung:	Highlights ► The effect of sodium intake on the outcome after myocardial infarction is unknown. ► High sodium intake favors inflammation and ventricular remodeling. ► High sodium intakers have higher short and long-term mortality after myocardial infarction.
ISSN:	0021-9150 1879-1484
DOI:	10.1016/j.atherosclerosis.2012.02.037